Does Infection Play a Role in Atherogenesis and Plaque Rupture?
动脉硬化与斑块破裂：感染是否在起作用？
Thus we now turn our attention to contemporary theories of atherogenesis.
因此，我们现在将注意力放在当代的动脉粥样化形成理论上。
Hemodynamic factors have been suggested to be important in atherogenesis.
普遍的观点认为血流动力学因素在其中起着重要作用。
Conclusion: BRPA has antioxidative and hypolipidemic effects, which make it reduce the risk of atherogenesis.
结论：BRPA具有降血脂和抗氧化等生物活性，可以降低动脉粥样硬化发生的危险性。
Objective To explore the relationship between serum bilirubin level and severity of coronary atherogenesis and serum lipid.
目的探讨血清胆红素水平与冠状动脉狭窄病变程度及血脂的关系。
Although some data from experimental models support a role of CRP in atherogenesis, its direct involvement remains in question.
尽管由实验模型得到的数据支持CRP在动脉粥样硬化形成中的作用，但它的直接关系仍在讨论中。
Conclusions - These results are the first to show that MBL is abundantly present and locally produced during early atherogenesis.
结论：这些结果首次提示MBL在动脉粥样硬化早期局域性大量表达。
Conclusion BRF has dramatically reduced serum anti ox LDL antibody levels and this effect may be related to its anti atherogenesis.
这表明黑米皮的抗动脉粥样硬化作用可能与其降低抗氧化型低密度脂蛋白抗体水平有关。
There is increasing evidence that lipid peroxidation and oxidative modification of low density lipoprotein is important in atherogenesis.
脂质过氧化和低密度脂蛋白的氧化修饰在动脉硬化的发生中起着极其重要的作用。
During atherogenesis, vascular smooth muscle cells proliferate, migrate from tunica intima to media and uptake lipid then become foam cells.
我们推测动脉粥样硬化形成过程中血管平滑肌细胞能量代谢发生了改变。
In bitter melon seeds, CLA may be one of the functional components related to the function of anticancer, anti-atherogenesis, antibacterial, anti-virus or regulation of immunity.
共轭亚油酸可能是苦瓜籽调节免疫、抗癌、抗动脉粥样硬化等作用的功能成分之一。
We hypothesized that PARP may play an important role in atherogenesis and that its inhibition may attenuate atherosclerotic plaque development in an experimental model of atherosclerosis.
我们假设PARP可能在动脉粥样硬化形成中起重要作用，它的抑制可能会减弱动脉粥样硬化实验模型中动脉硬化斑块的进展。
We hypothesized that PARP may play an important role in atherogenesis and that its inhibition may attenuate atherosclerotic plaque development in an experimental model of atherosclerosis.
我们假设PARP可能在动脉粥样硬化形成中起重要作用，它的抑制可能会减弱动脉粥样硬化实验模型中动脉硬化斑块的进展。
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