兴奋毒性可能是造成神经元死亡的“最后公路”。
The excitotoxicity may be the last public way by which neurons were led to death.
目的：探讨麝香酮减轻兴奋毒性、抗急性脑缺血损伤的作用。
Objective: To study the effect of muscone on attenuating excitotoxity and brain damage in acute cerebral ischemia.
结论：低温对脑缺血的保护机制之一可能是降低了神经细胞的兴奋毒性指数。
Conclusion: the neuroprotective effect of hypothermia on cerebral ischemia may reside, in part, in its ability to decrease extracellular excitotoxic indexes.
目的：研究N甲基D天冬氨酸（NMDA）诱导大鼠小脑颗粒神经元兴奋毒性保护作用的机制。
Objective:To study the mechanisms underlying N methyl D aspartate (NMDA) induced excitoprotection in cultured rat cerebellar granule neurons.
雌激素能拮抗兴奋性氨基酸毒性，稳定细胞内钙离子溶度。
Estrogen can resist the excitability amino acid toxicity and stable the solubility of calcium ion in cells.
结论黄芩苷对大鼠脑片缺氧缺糖性损伤具有一定的保护作用，作用机制可能与其抑制了兴奋性氨基酸毒性有关。
Conclusion Baicalin possesses protective effect on OGD injury in rat brain slices, which is related to its inhibitory effect on excitatory amino acids toxicity.
目的探讨兴奋性氨基酸受体拮抗剂磷酸甲基谷氨酰氨酸（GAPA）对胆红素神经毒性的影响。
Objective To explore the effect of NMDA receptor antagonist glutamyl amino methyl phosphonic acid (GAPA) on the bilirubin neurotoxicity.
结论：癫痫急性发作期n 1、P 1波的改变与兴奋性氨基酸的神经元毒性作用及继发性缺血、缺氧致神经元损伤有关。
Conclusion: the changes of N1P1 in acute attack period are related with the neuron toxic action of excitatory amino acids and lesions made by secondary ischemia and hypoxia.
因谷氨酸代谢异常，过度释放和对突触后受体的高频刺激，引起了兴奋性神经毒性，导致神经元死亡。
The abnormal metabolism and excess release of Glutamic acid as well as high-frequency stimulation to the postsynaptic receptor induce excitatory neural toxicity, resulting in the death of neurons.
我到亚利桑那州超过10次，但这是第一次看到美国西南沙漠地方的大毒蜥蝪，一种毒性很强的生物，看到它我们都很兴奋，因为它是非常稀有的。
I have been to Arizona over 10 times and this was the first time that I saw the Gila Monster lizard. A poisonous creature. We were all very thrilled to see it in the wilderness since it is very rare.
结论复方丹参片降低兴奋性氨基酸含量，以对抗兴奋性氨基酸的毒性，这可能是其防治ad的机制之一。
Conclusion CDST could lower the toxicity of excitatory amino acid by decreasing its contents, which may be one of the mechanisms of CDST in prevention and treatment to AD.
为评估虾青素对中风的保护，将测试脑组织被自由基损害、凋亡和兴奋剂毒性。
To evaluate the protective mechanisms of ATX against stroke, brain tissues were assayed for free radical damage, apoptosis, and excitoxicity.
为评估虾青素对中风的保护，将测试脑组织被自由基损害、凋亡和兴奋剂毒性。
To evaluate the protective mechanisms of ATX against stroke, brain tissues were assayed for free radical damage, apoptosis, and excitoxicity.
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