目的：克隆全反式维甲酸诱导前列腺癌细胞系DU 145细胞产生凋亡的相关基因。
Objective To clone the apoptosis-related genes induced by all trans-retinoic acid(ATRA) from prostatic carcinoma cell line DU-145, and investigate the molecular mechanism of the apoptotic process.
分离高转移性和低转移性前列腺癌细胞，筛选转移相关基因，为研究前列腺癌的转移分子标志奠定基础。
To isolate high metastatic and low metastatic prostate cancer cells and screen metastasis-associated genes for the purpose of the biomarker exploration of prostate cancer metastasis.
日前，约翰·霍普金斯的研究者发现在内分泌治疗无反应的患者的前列腺癌细胞的激素受体存在明显的差异。
Now, researchers at Johns Hopkins have discovered critical differences in the hormone receptors on prostate cancer cells in patients who no longer respond to this therapy.
结论：MAPK通路的持续活化在前列腺癌的恶性进展中起重要作用，阻断此通路可以抑制前列腺癌细胞的增殖。
Conclusion: The MAPK transpass activity is promoted during the prostate cancer progression, suggesting that blocking the MAPK transpass can be used as a method for prostate cancer therapy.
伦敦圣乔治大学的科学家们正在研究一种血液测试，即通过DNA标记物来识别出那些脱落进入血流当中的前列腺癌细胞。
Scientists at st George's, University of London, are working on a blood test that USES DNA markers to identify prostate cancer cells that are shed into the bloodstream.
为了弄清ar -V7的雄激素受体区别与其他，研究者发现实验室生长的前列腺癌细胞只产生AR - V 7序列。
To see how androgen receptors made from AR-V7 differ from others, the researchers forced lab-grown prostate cancer cells to produce only the AR-V7 sequence.
伦敦圣乔治大学的科学家们正在研究一种血液测试，即通过DNA标记物来识别出那些脱落进入血流当中的前列腺癌细胞。
Scientists at st George's, Uniersity of London, are working on a blood test that USES DNA markers to identify prostate cancer cells that are shed into the bloodstream.
此项研究的目的是揭示前列腺癌细胞抵制降低男性激素(如睾酮)治疗的机理，而细胞的生存正常情况下是需要男性激素的。
The goal of the research was to uncover how prostate cancer cells become resistant to treatment that lowers levels of male hormones such as testosterone, which the cells normally need to survive.
研究人员发现了新的小分子，这些小分子可能会在前列腺癌细胞将正常细胞转变为癌细胞过程中阻断癌细胞关闭正常基因的表达。
Researchers have discovered new small molecules that may prevent prostate cancer cells from turning off normal genes in a process that transforms normal cells into cancer cells.
目的：利用RNA干涉技术抑制雄激素受体（AR）的表达，研究AR在激素依赖性和激素非依赖性前列腺癌细胞增殖中的作用。
AIM: To study the role of androgen receptor (AR) in hormone-dependent and hormone-independent prostate cancer cell proliferation by knocking down AR expression with adenovirus-delivered siRNA.
RTVP - 1基因导致前列腺癌细胞发生细胞凋亡或称程序性细胞死亡，RTVP - 1基因也激活免疫系统攻击癌细胞。
It causes what is known as "apoptosis" or programmed cell death in prostate cancer cells and also activates the immune system to fight cancer cells.
目的：研究前列腺癌进展中细胞丝裂原活化蛋白激酶（MAPK）信号通路的变化，探讨阻断此通路对前列腺癌细胞增殖的影响。
To investigate the change of mitogen activated protein kinase(MAPK) signaling pathway during prostate cancer progression, and therefore to explore its role on cell proliferation.
“在我们的实验中，这种复合物在动物实验和实验室研究中均能抑制前列腺癌细胞的生长，”研究者RebeckaHellsten说。
"In our trials this compound has curbed the growth of prostate cancer cells both in animal experiments and in laboratory experiments, " says the researcher Rebecka Hellsten.
华盛顿(法新社)- - -周一的一个研究显示，美国研究者发现一种抗体可以追踪小鼠的前列腺癌细胞，并可以攻克这种到晚期可以致命的疾病。
WASHINGTON (AFP) - US researchers have found an antibody that hunts down prostate cancer cells in mice and can destroy the killer disease even in an advanced stage, a study showed Monday.
然后，研究小组在对老鼠的试验中显示，使用STO 609后，CAMKK2的产生和活性都被停止了，有效的堵塞了前列腺癌细胞的传播，并减小了肿瘤的大小。
The team then showed in mice that stopping both the production and activity of CAMKK2 using sto 609, blocked the spread of prostate cancer cells and reduced the tumour size.
这种疗法昂贵的部分原因在于，它不是一种实验室研制的药物，而是针对个体需要而采取的特定疗法，需要用到每位患者的细胞，以及大多数前列腺癌细胞所具有的蛋白质。
Part of why it costs so much is that it's not a pill cranked out in a lab, but a treatment that is individually prepared, using each patient's cells and a protein found on most prostate cancer cells.
目的：培养建立中国人种前列腺癌细胞系，为了解中国人种前列腺癌细胞生长特性、进一步开展前列腺癌基因诊断和药物筛选建立方便的实验手段和模型打下基础。
Objective: To culture and establish Chinese ethnic prostate cancer cell line, investigate its growth characterization and explore experimental methods and models of gene diagnosis and trial of drugs.
Griffith说，“我们的理想目标是希望能在总结这个试验时这样说，这是一种新的安全的疗法，能够按照预期引发前列腺癌细胞死亡而对正常细胞没有损害。”
Ideally, we hope to be able to say at the conclusion of this trial that this novel agent is safe and performs as intended by causing the death of prostate tumor cells with no harm to normal cells.
当在实验室中用该产品在肿瘤细胞中进行试验时，他们发现与单独使用睾酮相比，该产品能更有力的促进前列腺癌细胞的生长。他们在《临床肿瘤研究》杂志上报道了这一研究。
When they tested it on tumor cells in the lab, they found it fueled the growth of prostate cancer cells more potently than testosterone alone, they reported in the journal Clinical cancer Research.
对于前列腺癌的晚期，使用激素疗法会很有效，而且通常可以数年时间内有效地控制癌细胞的扩散。
Prostate cancer can also be treated with hormonal therapy in more advanced stages and this can be very effective and can often control the disease for many years.
也见于老年人的前列腺癌可致死，但由于其生长缓慢，所以多数病人在癌细胞扩散前即因其它原因而死亡。
Prostate cancer, also found mainly in older men, can be deadly but usually grows so slowly that most patients die of something else before the cancer spreads.
也见于老年人的前列腺癌可致死，但由于其生长缓慢，所以多数病人在癌细胞扩散前即因其它原因而死亡。
Prostate cancer, also found mainly in older men, can be deadly but usually grows so slowly that most patients die of something else before the cancer spreads.
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