目的构建大鼠重症急性胰腺炎(SAP)模型，探讨复合乳酸菌对SAP大鼠细胞因子和肠道免疫屏障的影响。
Objective to investigate the effect of compound lactobacilli on cytokines balance and intestinal immuno-barrier in established rats models of severe acute pancreatitis (SAP).
目的观察重症急性胰腺炎（SAP）大鼠白血病抑制因子（LIF）在胰腺组织中表达的时相变化，探讨LIF在SAP病程中的意义。
Objective To observe the phase changes in the expression of leukemia inhibitory factor(LIF) in pancreatic tissues in rats with severe acute pancreatitis(SAP) and the significance.
目的观察经空肠输注大黄煎液对重症急性胰腺炎大鼠肠道屏障功能的保护作用。
Objective To observe the effect of Rhubarb on the intestinal barrier of rats with severe acute pancreatitis (SAP).
结论免疫增强型肠内营养能有效调节重症急性胰腺炎大鼠细胞因子水平，增强免疫功能，缩短病程，降低死亡率。
Conclusion Immune-enhancing enteral nutrition can reduce cytokines, strengthen immune function, shorten disease course, and decrease the mortality in rats with severe acute pancreatitis.
目的观察黄芩甙和奥曲肽对重症急性胰腺炎(SAP)大鼠肺损害的影响，探讨两药对SAP的治疗作用与机制。
Objective To observe the influence of Baicalin and Octreotide on lung injury of rat with severe acute pancreatitis (SAP) and discuss the therapeutic effect and mechanism of the two medicines on SAP.
目的探讨胰炎灵颗粒剂对重症急性胰腺炎(SAP)模型大鼠的治疗作用及机理。
Objective To investigate the effects and mechanisms of Yiyanling granule on experimental severe acute pancreatitis (SAP) in Rats.
目的观察重症急性坏死性胰腺炎（SAP）大鼠肠壁、肝脏和肺组织中免疫单核吞噬细胞分布的变化，并探讨谷氨酰胺对其的调节作用。
Objective To observe the changes of immunocytes and macrophages in gut, liver and lung in rats with severe acute pancreatitis (SAP) and the effect of glutamine on these changes.
目的：观察重症急性胰腺炎(SAP)大鼠的脑损伤以及血脑屏障在SAP大鼠脑损伤中的作用。
Aim: to observe the brain damage in severe acute pancreatitis (SAP), and to explore the prevention brain from impairing treated by somatostatin combined with growth hormone in SAP rats.
方法： 分别采用腹腔注射雨蛙肽和胆胰管逆行注射5%牛磺胆酸钠建立大鼠轻症、重症急性胰腺炎模型。
Methods: Acute pancreatitis model was induced by intraperitoneal injection of cerulein or retrograde infusion of 5% sodium taurocholate into the bilipancreatic duct in SD rats.
方法： 分别采用腹腔注射雨蛙肽和胆胰管逆行注射5%牛磺胆酸钠建立大鼠轻症、重症急性胰腺炎模型。
Methods: Acute pancreatitis model was induced by intraperitoneal injection of cerulein or retrograde infusion of 5% sodium taurocholate into the bilipancreatic duct in SD rats.
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