目的探讨慢性缺氧引起缺氧性肺血管收缩反应降低的机制。
Objective To investigate the mechanism of chronic hypoxia induced decreased HPV.
布洛芬抑制前列腺素的生成，并通过增强缺氧性肺血管收缩反应而改善了肺损伤时的气体交换。
The inhibition of prostaglandin production with Ibuprofen strengthened hypoxic pulmonary vasoconstriction thus improved pulmonary gas exchanges in acid lung injury.
结果提示：高原移居平原者与世居平原者相比，脑血管缺氧性扩张反应较强，而肺血管缺氧性收缩反应较弱。
This study suggests that long term residing at high altitude results in an increase in cerebral vasodilative response and a decrease in pulmonary vasoconstrictive response to acute hypoxia.
缺氧时肺动脉压显著升高，这可能是肺毛细血管前肌性小动脉收缩的结果。
Hypoxia produced a sustained increase in pulmonary arterial pressure (Ppa), the site of vasoconstriction was proximal to the lung capillaries.
缺氧时肺动脉压显著升高，这可能是肺毛细血管前肌性小动脉收缩的结果。
Hypoxia produced a sustained increase in pulmonary arterial pressure (Ppa), the site of vasoconstriction was proximal to the lung capillaries.
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