在过去的十年中，过氧化物酶体蛋白易位的机理一直是研究的主题。
The mechanism of peroxisomal protein translocation has been the subject of vigorous research in the past decade.
不正常的过氧化物酶体是一种可以导致疾病和衰老的细胞体。
Peroxisomes are cell organelles that can cause disease and aging processes if they do not function properly.
过氧化物酶体主要存在于肝胰腺细胞和触角腺的迷路上皮细胞中。
The results showed that the peroxisome mainly exists in the R-cell of hepatopancreas and the labyrinth epithelium of antennary gland.
罗格列酮属于称为过氧化物酶体活化受体（PPAR）促效剂一类药物。
Rosiglitazone belongs to a class of drugs known as peroxisome proliferator-actiated receptor (PPAR) agonists.
过氧化物酶体是细胞的履行其他各种功能，包括过氧化氢代谢和氧化的不饱和脂肪酸。
Peroxisomes are organelles that perform a variety of functions, including the metabolism of hydrogen peroxide and the oxidation of fatty acids. Peroxisomes do not possess organellar DNA;
化疗药物联合过氧化物酶体增殖物激活受体(PPAR)的配体能够协同抑制肺细胞癌及卵巢癌细胞。
Chemotherapy given in combination with ligands for the peroxisome proliferator-activated receptor - (PPAR) synergistically inhibits the growth of lung and ovarian cancer cell lines.
研究过氧化物酶体增殖剂活化受体在脂毒性心肌病中的作用，可以为预防和治疗脂毒性心肌病提供新思路。
Studies of the role of the peroxisome proliferators-activated receptor plays in lipotoxic cardiomyopathy will provide new strategies to prevent and cure lipotoxic cardiomyopathy.
例如，后代身上出现的一种称为过氧化物酶体增殖物激活型受体 (Ppara)的基因对调节胆固醇和脂类在肝脏中进行转换起着至关重要的作用。
One gene that changed in offspring, for example — known as Ppara — is essential in cholesterol management and the liver's role in converting lipids.
例如，后代身上出现的一种称为过氧化物酶体增殖物激活型受体 (Ppara)的基因对调节胆固醇和脂类在肝脏中进行转换起着至关重要的作用。
One gene that changed in offspring, for example — known as Ppara — is essential in cholesterol management and the liver's role in converting lipids.
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