The causes of degeneration and death of the SGN in acoustic trauma are as yet not completely known.
噪声通过机械、代谢和血运等途径损伤毛细胞的机制已基本明确，但SGN的变性死亡的原因还不清楚。
This is an experimental research into acute acoustic trauma free entirely from any explosive factor.
这是一次完全剔除爆震因素的急性声创伤实验研究。
There was a possibility of acoustic trauma in the artillerymen with late—onset tinnitus or deafness.
晚期出现耳聋、耳鸣症状的炮兵，有听力损伤的潜在可能。
Conclusion: Prophylactic inhalation of negative air ions can increase the cochlear blood flow and promote microcirculation thus protecting the cochlea from acoustic trauma.
结论：预防性吸入空气负离子可增加耳蜗血流量，改善微循环，因而对保护噪声性耳蜗损伤是有益的。
Objective: to observe the protective effect of sound preconditioning on hearing in acoustic trauma and explore the relevant mechanism with regard to free oxygen radicals in the cochlea.
目的：观察声预处理对噪声性听力损失的保护作用，并从氧自由基途径探讨其机制。
Objective: to observe the protective effect of sound preconditioning on hearing in acoustic trauma and explore the relevant mechanism with regard to free oxygen radicals in the cochlea.
目的：观察声预处理对噪声性听力损失的保护作用，并从氧自由基途径探讨其机制。
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