Aim: To investigate the effect of hyperbaric oxygen (HBO) on vascular permeability after acute focal cerebral ischemia and reperfusion in rats.
目的：探讨高压氧(HBO)对大鼠急性局灶性脑缺血再灌注损伤血管通透性的影响。
Objective To study whether nitric oxide (NO) and neuronal NO synthase (nNOS) were involved in the pathogenesis of acute focal cerebral ischemia and reperfusion.
目的探讨一氧化氮(NO)和神经元型NO合酶(n NOS)是否参与急性局灶性脑缺血再灌注的发病机理。
Conclusion The results suggest that neuron membrane structural component have changed during acute focal cerebral ischemia but GM1 might be more resistant to ischemia.
结论脑缺血可以导致神经质膜结构成分发生变化而神经节苷脂gm 1对脑缺血具有脑保护作用。
Methods The acute transient focal cerebral ischemia-reperfusion model was established by modified thread-occlusion method.
方法采用改良线栓法建立大鼠局灶性脑缺血再灌注模型。
Methods The acute transient focal cerebral ischemia-reperfusion model was established by modified thread-occlusion method.
方法采用改良线栓法建立大鼠局灶性脑缺血再灌注模型。
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