Methods: An acute hepatic failure model was established in ducklings infected DHBV with lipopolysaccharides (LPS).
方法：用脂多糖(LPS)诱发DHBV感染雏鸭急性肝坏死，建立实验模型。
Conclusion: the mechanism of blood glucose regulation appears to be abnormal in the model of acute hepatic failure rats, and high dose of LPS may lead to hypoglycemia.
结论：急性肝功能衰竭的模型中有血糖调节机制的异常，大剂量的内毒素导致低血糖的发生。
Conclusion: the mechanism of blood glucose regulation appears to be abnormal in the model of acute hepatic failure rats, and high dose of LPS may lead to hypoglycemia.
结论：急性肝功能衰竭的模型中有血糖调节机制的异常，大剂量的内毒素导致低血糖的发生。
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