Conclusion: the expression of AQP1 was increased in adenocarcinoma of the lung over normal lung tissues.
结论：AQP1在肺腺癌组织中表达增高，在正常组织中较低。
The passive smoking in nonsmokers was a main oncogenesis in lung cancer. Adenocarcinoma of the lung was possibly induced by environmental tobacco smoke (ETS).
在非吸烟者中，被动吸烟是增加患肺癌的一种重要因素。在细胞组织学类型上其主要诱发腺癌。
AIM: to study the apoptosis of human lung adenocarcinoma A549 cells induced by CN-2, a derivative of podophyllotoxin, and to obtain insights into its molecular mechanism of action.
目的研究鬼臼素衍生物cn - 2对人肺腺癌细胞A549的凋亡诱导作用，并对其分子机制进行初步探讨。
Conclusion The overexpression of CC10 might play an important role in the matrine-induced apoptosis of lung adenocarcinoma A549 cells.
结论CC10基因表达增高可能在苦参碱诱导肺腺癌A549细胞凋亡中起重要作用。
Objective To investigate the chemosensitivity of irradiated resistant cells of multicellular spheroids in A549 lung adenocarcinoma.
目的探讨A549肺腺癌多细胞球体放射抗拒细胞的化疗药物敏感性。
ObjectiveTo study the abnormity and clinical value of tyrosine kinase signaling pathway in lung adenocarcinoma.
目的探讨肺腺癌患者酪氨酸激酶传导通路的异常和临床预后的关系。
This research aims to study the growth characteristics of A549 lung adenocarcinoma MTS so as to choose the best ones to perform experiments.
本研究旨在了解A549肺腺癌MTS的生长特性，有助于选择最佳MTS用于实验研究。
Objective To study the effect of MVP chemotherapy combined with radiotherapy for advanced lung adenocarcinoma.
目的探讨化疗联合放疗对晚期肺腺癌预后的影响。
Adenocarcinoma is the one cell type of primary lung tumor that occurs more often in non-smokers and in smokers who have quit.
腺癌是单细胞类型的原发肺癌，多发生于非吸烟者和已戒烟者。
Objective To study the effect of GSSG modified superparamagnetic particles on the growth of human lung adenocarcinoma cell line SPC-A1 in vitro.
目的研究氧化型谷胱甘肽(GSSG)修饰的超顺磁性纳米粒子在体外对人肺腺癌细胞SPC - A 1生长的影响。
Smoking and passive smoking are among the causative factors of lung cancer in women, mainly of peripheral adenocarcinoma.
女性肺癌以周围型腺癌多见、发病年龄早、转移早、预后差、痰找肿瘤细胞是诊断肺癌的方法之一。
Objective: To investigate the chromosome and gene alterations in adenocarcinoma of lung cell line, om.
目的：研究分析肺腺癌细胞系OM的染色体及基因异常变化。
To investigate the suppression effect of tumor suppressor gene in lung adenocarcinoma cells.
探讨肿瘤抑制基因对肺腺癌细胞生长的抑制作用。
Conclusion: Transfection of DTA gene in vitro may kill human lung adenocarcinoma cells and reduce the oncogenicity of human lung adenocarcinoma cells.
结论体外转染白喉毒素a基因可杀伤人肺腺癌细胞并降低人肺腺癌细胞的致瘤性。
Objective To study the correlation between tumor markers and CT characteristics of lung adenocarcinoma.
目的探讨肺腺癌CT 征象与肿瘤标记物的关系。
Objective to study the DNA damage and repair of normal lung interstitial cells and human lung adenocarcinoma cells exposed to cigarette smoke.
目的探讨经香烟烟雾溶液染毒的人正常肺间质细胞和人肺腺癌细胞的DNA损伤及其修复效应。
Conclusion Aspirin can inhibit the proliferation of lung adenocarcinoma cells A549 through influencing the distribution of cell cycle and inducing apoptosis of A549 cells.
结论阿司匹林可抑制肺腺癌细胞A549的增殖，其影响细胞周期分布、诱导细胞凋亡可能是其重要的机制。
To screen differential expression protein of human lung adenocarcinoma cell A549 treated with radiation and to discuss the role of these proteins.
筛选了人肺腺癌细胞A549放射前后的差异蛋白质，并探讨了其在放射中的作用。
CONCLUSION: Pharmacokinetic MRI proves to be a quantitative indicator for the response of brain metastasis to the radiotherapy for lung adenocarcinoma.
结论：药动力学MRI影像技术是有效评估放疗脑内肺腺癌转移性病灶的方法，可以为临床提供准确定量的依据。
Objective: To study the influence of N-acetylcysteine (NAC) on the growth and spontaneous metastasis of lung adenocarcinoma in T739 mice.
目的：探讨n -乙酰半胱氨酸(NAC)对T739小鼠肺腺癌原发瘤形成及自发性肺转移的影响。
The end stage of lung adenocarcinoma is often associated with distant metastasis and formation of malignant pleural effusion (PE).
肺腺癌早期症状不明显且不易察觉，但末期常并发远端转移及恶性肋膜腔积水。
Objective To examine the relationship between tumor-associated macrophages (TAMs) infiltration and the density of lymphangiogenesis or the patients' clinical outcome in lung adenocarcinoma.
目的探讨肺腺癌肿瘤相关巨噬细胞(TAM)浸润与淋巴管生成、患者生存的关系。
Objective: to study Somatic mutation of the tyrosine kinase (TK) domain of the epidermal growth factor receptor (EGFR gene) in lung adenocarcinoma patients.
目的：探讨表皮生长因子受体(EGFR)基因酪氨酸激酶域体细胞在肺腺癌患者中突变的相关因素。
Conclusions Metformin can inhibit the growth of lung adenocarcinoma cell lines significantly and potentiate cisplatin in a dose - and time-dependent manner in vitro.
二甲双胍与顺铂共同作用可以增强对肺腺癌细胞生长的抑制作用。
CONCLUSION: UGT1A7 gene polymorphisms may increase the genetic susceptibility of lung adenocarcinoma in Chinese.
结论：UGT1A7基因多态可能是中国人肺癌遗传易感性因素。
Besides, the cause of death related to pathologic typing, of 7 cases of death, lung adenocarcinoma, small cell and large cell carcinoma were 5 case in all, the squamous cell only 2 cases.
死亡原因与病理类型也有一定的关系，在死亡的7例中，腺癌、小细胞癌及大细胞癌占5例，鳞癌仅占2例。
The positive expression rate of CEA increased significantly in lung adenocarcinoma, and SCC and CA211 in lung squamous cell carcinoma and lung adenocarcinoma, while NSE in small cell lung cancer.
CEA在肺腺癌中表达阳性率明显增高；scc、CA211在肺鳞癌和腺癌中表达阳性率明显增高；nse在小细胞肺癌中表达阳性率明显增高。
ResultsNone of the 95 noncancerous tissues exhibited DNMT1 immunoreactivity, whereas 48 (50.5%) of the 95 lung adenocarcinoma cancerous tissues were DNMT1positive.
结果95例肺腺癌中有48例患者呈癌组织DNMT1阳性表达（50.5％），癌旁正常肺组织中DNMT1阴性表达；
Moreover, the unity of MMP-2 and VEGF immunocytochemistry is helpful for pathological diagnosis of adenocarcinoma cells of lung cancer.
两者联合做免疫细胞化学检查对肺腺癌细胞病理诊断有辅助意义。
ConclusionThere exists internal relations between the chemoresistance and radiosensitivity of lung adenocarcinoma cisplatin-resistant cell, chemo-resistance leads to reduced radiosensitivity.
结论肺腺癌细胞的化疗耐药性与放射敏感性之间有一定的内在联系，化疗耐药性导致放疗敏感性降低。
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