They discovered that amyloid beta binds to the prion proteins normally found on neurons.
研究人员发现与，淀粉样蛋白与通常是在神经元上发现的朊病毒蛋白结合在一起。
Those plaques contain billions of copies of amyloid beta all stuck together in one gloppy mess.
这些斑块含有数十亿的：-淀粉样蛋白拷贝，它们乱七八糟地堆积在一起形成一个糊状的大块。
One of the most promising is to inhibit an enzyme that shears off amyloid beta from a larger protein.
最优希望的一种就是抑制一种将β淀粉状蛋白从大蛋白中剪切下来的酶。
Amyloid beta is best known as the protein that forms the giant plaques that riddle the brains of people with Alzheimer's.
众所周知？ -淀粉样蛋白导致老年痴呆症患者的大脑里面形成巨大的斑块。这些斑块含有数十亿的。
The NYU researchers stitched together the string of amino acids on amyloid beta to create their synthetic peptide "mimic."
NYU的研究员将β淀粉状蛋白的氨基酸链整合以创造出模仿的合成肽。
Amyloid beta itself might be injuring nerve cells or the plaques, made of accumulations of amyloid beta, could be the culprits.
淀粉样β蛋白本身可能损伤神经细胞或者有它聚集形成的老年斑才是罪魁祸首。
One of the products, with the long-winded name amyloid beta-peptide 42, has the unfortunate property that its molecules like to stick to each other.
所形成的产物之一有一个冗长的名字——淀粉样贝塔-肽42，它具有一种不祥的特性，即它们分子彼此之间容易纠缠在一起。
Cells in the hippocampus are susceptible to damage caused by amyloid beta-derived diffusible ligands (ADDLs), toxic proteins that build up in people with Alzheimer's.
海马的细胞对淀粉贝塔·亚基(addls)造成的破坏敏感，ADDLS是造成阿尔兹·海默病的有毒蛋白质。
For the past decade there has been a lot of work on drugs called secretase inhibitors, which alter the activity of the enzymes that create amyloid beta-peptide 42.
在过去的10年里人们对被称为分泌酶抑制剂的药物进行了大量研究，这种药物可以改变酶类产生淀粉样贝塔-肽42的活动。
Traditional drug development in Alzheimer's disease is taking too narrow an approach by focusing intensively on the buildup of amyloid beta-protein in the brain, Dr. Nixon says.
传统的治疗老年痴呆症的药物研发思路仅仅集中在淀粉样β蛋白的累积上，Nixon博士说。
People with the disease get plaques in their brains made up of a small protein called amyloid-beta, which clumps together and disrupts brain signals.
得了这种病的人大脑里面的血小板形成一种叫做淀粉样蛋白的蛋白质，这种蛋白质聚集在一起打乱了大脑发出的信号。
Earlier work done by her team has shown that, in mice at least, the herpes infection of nerve cells induces accumulation of beta amyloid, the main component of amyloid plaques.
她的团队已经在早期做了大量的工作，至少在小鼠中，疱疹病毒感染的神经细胞导致了beta-淀粉的沉积，而这是淀粉样蛋白斑块的主要成分。
Amyloid plaques form when a molecule called amyloid precursor protein (APP) is chopped up by two enzymes known as beta-secretase and gamma-secretase.
当一种叫淀粉样前体蛋白（APP）的分子被两种酶——分别叫作贝塔-分泌酶和伽玛-分泌酶——切断时就形成了淀粉样斑块。
Research into new treatments for Alzheimer's, for example, has started to overturn what we thought we knew about the role of amyloid-beta proteins in the disease.
比如说，关于老年痴呆新疗法的研究已经开始颠覆我们之前淀粉状贝塔蛋白质的角色的认识。
He has tested it so far in mice, and found the animals have better memory and other cognitive skills and fewer clumps of beta-amyloid and tau protein.
目前的实验对象为老鼠。他还发现动物具有更强的记忆力和其他认知能力，更少的贝塔淀粉样和tau蛋白质凝结。
One of the telltale signs of this disease is the deposit of beta-amyloid plaques in the brain.
这种疾病的众多隐秘信号之一是在脑中的B -淀粉样蛋白斑块中的沉积。
A key aspect of Alzheimer's is sticky clumps of an abnormal protein in the brain called beta amyloid plaques.
阿尔兹·海默氏症主要是由于脑部聚集结块的异常蛋白质，名为be ta淀粉状蛋白斑。
Studies with glucose are being supplemented by PET scans that can image beta-amyloid, one of the abnormal proteins in the brain in Alzheimer’s disease.
对葡萄糖的研究加上PET扫描就能够对β-淀粉样蛋白成像，这是老年痴呆症病人大脑中一种异常的蛋白质。
It contains antibodies against a protein called beta-amyloid, which many Alzheimer's researchers believe plays a pivotal role in the progression of the disease.
它包含能够对抗一种淀粉样蛋白(beta -amyloid)的抗体，许多阿滋海默症研究者都认为这种淀粉样蛋白在疾病的形成中扮演了关键的角色。
Beta (in the form of beta-amyloid plaques) and tau (in the form of tau tangles) are the physical manifestations of Alzheimer's.
贝塔(以贝塔-淀粉样斑块的形式)和陶(以陶蛋白缠结的形式)是痴呆症的物质表征。
The causes for Alzheimer's disease are not known, but dysregulation of amyloid-precursor protein expression and beta-amyloid clearance is hypothesized (beta-amyloid cascade).
虽然导致老年痴呆症的原因还没有发现，但是有人假设其原因是淀粉样前提蛋白的调节异常和β-淀粉样蛋白的消失（β-淀粉样蛋白连锁反应）。
By electron microscopy, amyloid is composed of a "beta-pleated sheet" of fibrils, seen here as irregular grey material.
电镜，淀粉样物质是由原纤维的β-折叠组成。可见不规则的灰色物质。
Enzymatic digestion of Bence Jones proteins in vitro has shown that typical amyloid fibrils with a beta-pleated sheet configuration can be formed from some, but not all, Bence Jones proteins.
本周蛋白在体外的酶消化过程显示典型的具有β-折叠片结构的淀粉样蛋白纤维能通过一些，但不是全部的，本周蛋白生成。
If a person's immune system isn't up to clearing beta-amyloid, perhaps a boost with antibodies, such as those in Gammagard, would help.
如果一个人的免疫系统不能胜任清除贝塔-淀粉样物质，例如象那些在Gammagard中的抗体的促进或许能有所帮助。
If a person's immune system isn't up to clearing beta-amyloid, perhaps a boost with antibodies, such as those in Gammagard, would help.
如果一个人的免疫系统不能胜任清除贝塔-淀粉样物质，例如象那些在Gammagard中的抗体的促进或许能有所帮助。
应用推荐