Conclusions Madopar has a significant inhibitory effect on the apoptosis of neuron cells of rats with PD.
结论美多芭对帕金森病大鼠神经元细胞凋亡有明显抑制作用。
Conclusion: Cerebrolysin has the protection and treatment effect on HIE. The mechanism of the effect is correlated with blocking abnormal metabolism of no and reducing apoptosis of neuron.
结论：脑活素对HIE具有防治作用，其机理与阻断NO异常代谢、降低细胞凋亡有关。
Objective to explore the influences of early interventions on learning, memory and neuron apoptosis in the brain of neonatal rats with hypoxic ischemic brain damage (HIBD).
目的探讨早期干预对缺氧缺血性脑损伤(HIBD)大鼠学习记忆能力及神经元凋亡的影响。
Conclusion the expressions of STAT3 increased might indicate the transmission course in ischemic nerve cell signal, and play a key role in neuron apoptosis of ischemic brain injury.
结论STAT 3蛋白表达增强可能介导缺血性脑损伤的信号转导过程，其在脑缺血损伤神经元凋亡过程中起关键作用。
Objective to explore the effect of Tongxinluo capsules on cortex angiogenesis and neuron apoptosis within focal cerebral ischemic rat brain.
目的：探讨通心络胶囊对大鼠脑梗死皮层血管新生与神经元凋亡的影响。
Objective to investigate the effect of colony stimulating factor (CSF-1) on neuron survival and apoptosis in vitro.
目的观察集落刺激因子(CSF - 1)对神经元体外存活及凋亡的影响。
Objective to observe the role of mitochondria dysfunction in neuron apoptosis during ischemia-reperfusion injury.
目的观察线粒体功能失调在缺血再灌后神经元凋亡中的作用。
It is proved that current stimulation can promote regeneration of nerve, but it is scarcely reported on the effect of current stimulation on neuron apoptosis.
电刺激能有效促进神经的再生，但对神经元凋亡的影响研究较少。
Objective to study the influence of spinal cord decompression sickness on neuron apoptosis in rats and the protective effect of nerve growth factor.
目的探讨脊髓减压病对大鼠脊髓神经细胞凋亡的影响及神经生长因子的保护作用。
Objective: to observe the effect of excess iodine caused by supplying iodine during different gestation period on the neuron apoptosis and gene expression of filial rats.
目的：观察不同孕期补碘所引起的高碘对子鼠脑凋亡及调节基因表达的影响情况。
Conclusion: NOS inhibitors play a obvious protective role in long-term intelligence of fetal rat after delivery through relieved neuron apoptosis during hypoxic ischemia encephalopathy.
结论：NOS抑制剂通过减轻缺血缺氧脑损伤时胎鼠脑神经细胞凋亡对其出生后的远期智能起保护作用。
The pathophysiological mechanisms of ischemic cerebral injury involved in the release of excitatory amino acids, depolarization about the infarction, inflammatory reaction and neuron apoptosis.
缺血性脑损伤的病理生理机制包括兴奋性氨基酸的释放、梗死灶周围去极化、炎症反应及神经元凋亡等。
This suggests the diminished expression of NGF and impairment of neuron survival signal transduction pathway may be one of the reason of apoptosis in AD brains.
AD患者颚叶神经营养因子及其受体表达减少。神经营养因子表达的减少以及存活信号转导通路的损害可能是导致ad脑神经元凋亡的原因之一。
Conclusion the toxicity of lead on neuron development might related to the change of trace element concentration, reduction of antioxidation ability and increasing apoptosis in brain.
结论铅的发育神经毒性与中枢神经系统铜等微量元素的改变、抗氧化能力下降及由此所致的细胞凋亡数量增加有关。
RESULTS: Neuron necrosis, apoptosis, increase of apoptosis protein expression and proliferation of glial cells were the characteristics of pathological injury in the pilocarpine induced se models.
结果：毛果芸香碱惊厥持续状态模型鼠病理损伤特点为神经元坏死、细胞凋亡、凋亡蛋白表达增多，胶质细胞增生。
By literature reviewing, the majority of differential expression proteins have been reported to participate in neuron growth, apoptosis and stress-reaction.
结合文献复习，差异蛋白质主要涉及到神经细胞的增殖、凋亡、应激反应等过程。
Objective:To investigate the effect of elec-acupuncture on act and neuron apoptosis in substantia of Parkinson's disease in rats.
目的：研究电针对帕金森病模型大鼠黑质神经细胞凋亡和行为学的影响。
Relation between neuron apoptosis in hippocampus CA1 region and the pathogenesis of vascular dementia;
目的初步探讨海马CA1区神经细胞凋亡与丢失在血管性痴呆形成中的作用。
Objective: Perinatal hypoxic-ischemic brain injury is an important cause of neurodevelopmental impairment and disability. It's well-known that the main death 'form of neuron after HIBD is apoptosis.
目的：围产期缺氧缺血性脑损伤是导致儿童神经系统发育障碍及后遗症的严重疾病，已知脑损伤后迟发性神经元死亡以凋亡为主。
Objective: Perinatal hypoxic-ischemic brain injury is an important cause of neurodevelopmental impairment and disability. It's well-known that the main death 'form of neuron after HIBD is apoptosis.
目的：围产期缺氧缺血性脑损伤是导致儿童神经系统发育障碍及后遗症的严重疾病，已知脑损伤后迟发性神经元死亡以凋亡为主。
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