Calcium tends to build up in atherosclerotic plaques.
钙易于在动脉粥样斑块中堆积。
Background: Atherosclerotic plaques in the aortic arch are a risk factor for ischemic stroke.
研究背景：主动脉弓处动脉粥样斑块是缺血性卒中的危险因素。
Carotid atherosclerotic plaques were defined by cervical vascular color ultrasonic inspection.
颈动脉粥样硬化斑块经颈部血管彩色超声检查确定。
In recent years, the need for a more detailed analysis of atherosclerotic plaques has been stressed.
在近年，对一个动脉粥状硬化的饰板的更详细的分析的需要已经被强调。
Atherosclerosis is a systemic inflammatory disease characterized by the formation of atherosclerotic plaques.
动脉粥样硬化症是以动脉粥样硬化斑块形成为特征的全身性炎症性疾病。
In some patients, this may alter atherosclerotic plaques and cause clots in coronary arteries leading to heart attacks.
对某些病人，它可以使动脉粥样硬化斑块破裂形成冠状动脉血栓以致心脏病。
The immunohistochemistry analysis was used to measure the expression of ICAM-1 in rabbit aortic atherosclerotic plaques.
采用免疫组织化学分析ICAM-1在主动脉粥样斑块中的表达量。
The instability of atherosclerotic plaques is the important reason that may result in plaque rupture and then cause stroke.
动脉粥样硬化斑块不稳定是导致斑块破裂进而引起卒中的重要原因。
The carotid atherosclerotic plaques in patients with embolus detachment had more obvious inflammatory cellular infiltration.
有栓子脱落患者的颈动脉粥样硬化斑块具有更明显的炎性细胞浸润。
Results:The rabbit plaque in situ hybridization revealed that the gene fragment of atherosclerotic plaques in tissue proliferation.
结果：兔斑块组织原位杂交显示该基因片段在动脉粥样硬化斑块组织中表达水平增高。
Carotid atherosclerosis is a common cause of cerebral infarction and the stability of carotid atherosclerotic plaques is the key factor.
颈动脉粥样硬化是导致脑梗死的常见原因，其中颈动脉粥样硬化斑块的稳定性起着决定性作用。
International Circulation: Both high levels of LDLc and low levels of HDLc play key roles in the progression of atherosclerotic plaques.
《国际循环》：高ldl - C和低hdl - C在动脉粥样硬化斑块的进展中均发挥重要作用。
Losartan treatment can obviously decrease the plasma level of inflammatory factors, and it is good for the stability of atherosclerotic plaques.
氯沙坦治疗可以明显降低血浆炎症因子水平，有利于动脉粥样硬化斑块稳定。
Results Atherosclerotic plaques and extensive lipid infiltration appeared in myocardium tissue and cell matrix of hyperlipidemia animals separately.
结果高脂组心肌组织中有粥样硬化斑块的形成，间质内有大量脂质沉积、浸润。
The results showed that the size of atherosclerotic plaques and TC, HDL-CH, LDL-CH, HDL-PL, TG and blood viscosity were also significantly decreased.
结果表明，脂质体组主动脉内膜粥样硬化斑块面积、血脂各项指标及血液流变学等均显著改善。
The instability of atherosclerotic plaques is the leading pathogenesis of stroke resulting in plaque rupture and ulcer-caused thrombosis and it is very harmful.
动脉粥样硬化斑块的不稳定是导致斑块破裂、溃疡致使动脉内血栓形成从而引起卒中的主要发病机制，具有极大的危害性。
These temporal phenotypic plaque alterations should be taken into account for biomarker and therapeutic target validation studies using human atherosclerotic plaques.
在使用人类粥样硬化斑块作为生物标志物和治疗标靶的有效性研究中，应该考虑这些暂时性的斑块类型的改变。
Atherosclerotic plaques of cholesterol mainly from LDL, LDL-C can be used as evaluation of the prognosis of patients with stable angina objective material indicators.
动脉粥样硬化斑块中的胆固醇主要来自LDL,LDL - C可以作为评价稳定型心绞痛患者预后的客观物质指标。
The mural thrombi coming off from the unstable, atherosclerotic plaques may serve as the sources of small emboli and may also be one of the causes of cerebral infarction.
不稳定的颈动脉粥样硬化导致的斑块脱落可能也是脑梗死的病因之一。
Recent studies have suggested that vascular endothelial growth factor, matrix metalloproteinase-2 and-8 play important roles in the instability of atherosclerotic plaques.
近年来的研究表明，血管内皮生长因子、基质金属蛋白酶- 2和基质金属蛋白酶- 8在动脉粥样硬化斑块不稳定中起着重要作用。
The severity of stenosis of the extracranial carotid and the formation of the atherosclerotic plaques were significantly correlated to the risk factors of cerebrovascular disease.
斑块发生率及颅外段颈动脉狭窄程度与脑血管病危险因素有明显相关性。
Conclusion HO-1 inhibits progression of advanced atherosclerotic plaques and promotes plaque stability, probably by modulating plaque composition and attenuating plaque inflammation.
结论HO - 1可抑制动脉粥样硬化进展，增加斑块的稳定性，其机制可能与调节斑块结构和成分，抑制斑块内炎症有关。
Objective To observe the blood lipid level changes in patients with different types of carotid atherosclerotic plaques and the correlation between blood lipid level and plaque stability.
目的观察不同类型颈动脉粥样硬化斑块患者血脂水平的变化及与斑块稳定性的相关性。
Tissue factor (TF) is found in the adventitia of blood vessels and in the lipid core of atherosclerotic plaques, and is specifically expressed on monocyte or macrophage cell membrane surfaces.
研究发现组织因子(TF)位于血管外膜和粥样斑块的脂质中心，并特异地表达于单核细胞和巨噬细胞膜表面。
Evidence continues to accumulate associating severe periodontitis with an increased risk of forming atherosclerotic plaques, which are responsible for myocardial infarction and ischemic stroke.
越来越多证据显示，严重的牙周炎可促进形成动脉粥样硬化斑块。动脉粥样硬化斑块的形成是心肌梗塞和缺血性中风的成因。
Objective To study the expression of plasminogen activator inhibitor-1(PAI-1) of the blood serum and plaques in atherosclerotic rabbits in order to explore the effect of PAI-1 on atherosclerosis (AS).
目的通过对动脉粥样硬化（AS）兔血清和粥样硬化斑块中纤溶酶原激活物抑制物-1（PAI-1）表达的研究，探讨PAI-1在AS中的作用。
Plaques or atherosclerotic lesions, are fatty, inflamed deposits that form on the inside walls of arteries.
斑块或粥样硬化病变是指形成动脉壁内层的脂肪炎性沉着物。
Plaques or atherosclerotic lesions, are fatty, inflamed deposits that form on the inside walls of arteries.
斑块或粥样硬化病变是指形成动脉壁内层的脂肪炎性沉着物。
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