Conclusion: SEP is the useful modality for monitoring brain ischemia.
结论SEP是脑缺血的有用指标。
It suggests that chronic brain ischemia affects rat spatial learning and memory.
说明慢性脑缺血影响了大鼠的空间学习记忆。
METHODS: Thread-bolt occlusion method was used to establish focal brain ischemia model on rats.
方法：采用线栓并环扎的方法建立大鼠局灶脑缺血模型。
Glumate (Glu) released from neurons during brain ischemia is a main cause of inducing neuron death.
脑缺血时，神经元释放的谷氨酸是引起神经元死亡的重要原因。
Conclusion the methods can successfully establish the brain ischemia rat model combining blood stasis.
结论采用上述方法可以成功建立血瘀性脑缺血大鼠模型。
Methods The model of transient brain ischemia-reperfusion was established in gerbils by bilateral carotid artery clamping.
方法采用夹闭双侧颈总动脉的方法，制备沙土鼠短暂性全脑缺血-再灌注模型。
DWI is sensitive to early ischemic changes, which can find the changes of brain ischemia earlier than that of routine T2WI.
DWI对早期缺血改变非常敏感，DWI较常规t2加权图像能更早地发现脑缺血的改变。
Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.
结论：老龄大鼠脑缺血再灌注脑组织损伤与钙超载和自由基损伤有关。
This result supports the use of S100B as a surrogate marker for brain ischemia in patients with subarachnoid aneurysmal hemorrhage.
该结果支持了S100 B可作为蛛网膜下动脉瘤破裂出血患者脑缺血的代用标记物。
The purpose was to observe the changes of cerebral blood flow at different position using the model of the cats with brain ischemia.
应用猫局灶性脑缺血模型，利用计算机控制的激光多普勒血流计，持续监测猫脑不同位置在脑缺血时的血流变化。
Objective To explore the feasibility of neural stem cells (NSCs) transplantation for focal brain ischemia-reperfusion injury in rats.
目的探讨大鼠胚胎神经干细胞移植治疗局灶性脑缺血再灌注损伤的可行性。
METHODS: Acute global brain ischemia-reperfusion models were established in rats of puerarin treatment group and model control group.
方法：葛根素治疗组及模型对照组建立大鼠急性全脑缺血再灌注模型。
Objective To explore the protective of Naloxone hydrochloride on brain and its mechanism during complete brain ischemia-reperfusion injury.
目的探讨纳洛酮对全脑缺血再灌注损伤的防治作用及其机理。
Using the model of the rabbit brain ischemia the intracellular calcium accumulation and ultrastructure alteration of cerebral cortex were observed.
用兔全脑缺血模型，观察了缺血再灌注期脑组织钙积聚和超微结构改变。
Conclusions the brain ischemia reperfusion injury in young and the aged rats were correlated with the decrease of NO level and the increase of TNF.
结论青年大鼠和老龄大鼠脑缺血再灌注脑组织损伤与NO含量降低和TNF增高有关。
This phenomenon was called cerebral ischemic tolerance, and the sublethal brain ischemia given in advance was called cerebral ischemic preconditioning.
远端缺血预处理是指预先对靶器官以外的组织或器官进行缺血预处理，使靶器官产生缺血耐受的现象。
Conclusion DG can markedly inhibit nerve cell apoptosis induced by focal brain ischemia reperfusion injury and protect the nerve cell to a certain extent.
结论DG可明显抑制局灶性脑缺血-再灌注所诱导的脑神经细胞凋亡，起到一定的神经保护作用。
Conclusion: nimodipine and aspirin in treatment for brain arterial spasmodic headache and dizziness was not only more efficacious nimodipine, but also prevented brain ischemia.
结论尼莫地平加小剂量阿司匹林治疗脑血管痉挛性头痛头晕优于单用尼莫地平，并可预防缺血性脑卒中的发生。
In our study we investigated the influence of ginseng total saponin on brain tissue injury after ischemia-reperfusion by the model of brain ischemia-reperfusion with rat models.
本课题利用大鼠脑缺血再灌注模型，观察人参总皂甙对缺血再灌注后脑组织损伤的影响，目的是探讨人参总皂甙对大鼠缺血再灌注脑组织的保护作用机制。
Dexamethasone exacerbated ischemic neuronal injury but dexamethasone combined with mannitol was the most effective treatment for brain ischemia injury compared with other groups.
与其他处理组比较，地塞米松处理组缺血性脑损伤表现最严重，而在地塞米松复合甘露醇处理组脑损伤表现最轻。
Specifically, we will substantially study the therapeutic effect of ASCs on TBI-associated brain ischemia and inflammation via intravenous administration or by intro-cerebral transplantation.
具体地说，我们将大量研究的治疗效果携带者的照射有关的脑缺血和炎症通过静脉注射或介绍脑移植。
In roughly one-third of SAH victims, the blood vessels suddenly constrict, slowing the flow of oxygen-rich blood to the brain and causing a condition known as delayed ischemia.
大约有三分之一的sah患者的血管会突然收缩，减缓含有氧气的血液流向大脑的速度，造成一种称为延迟缺血的病情。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
It is true that acute ischemia can damage brain seriously.
证实了急性脑缺血对脑组织造成严重损害。
Results the Danhuang capsule can increase SOD AND Evans blue content of brain tissue from rat model of acute incomplete cerebral ischemia, at the same time, decrease the LPO content.
结果丹黄通络胶囊能提高急性不完全性脑缺血模型的大鼠脑组织中的SOD和伊文思兰的含量，降低LPO含量。
The results indicated the upward regulation of TR in rat brain during acute cerebral ischemia is a compensatory response, which allows the maximum binding of thyroid hormones.
大鼠脑缺血急性期脑甲状腺激素受体向上调节为机体的重要代偿反应。
RESULTS: in flunarizine therapy group, the degree of injury of brain tissue was significantly lighter than that at each time in cerebral ischemia group.
结果：氟桂嗪治疗组在再灌注各个时间点脑组织损伤程度均明显轻于脑缺血组。
RESULTS: in flunarizine therapy group, the degree of injury of brain tissue was significantly lighter than that at each time in cerebral ischemia group.
结果：氟桂嗪治疗组在再灌注各个时间点脑组织损伤程度均明显轻于脑缺血组。
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