Objective To explore the feasibility of neural stem cells (NSCs) transplantation for focal brain ischemia-reperfusion injury in rats.
目的探讨大鼠胚胎神经干细胞移植治疗局灶性脑缺血再灌注损伤的可行性。
Objective To explore the protective of Naloxone hydrochloride on brain and its mechanism during complete brain ischemia-reperfusion injury.
目的探讨纳洛酮对全脑缺血再灌注损伤的防治作用及其机理。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Conclusion DG can markedly inhibit nerve cell apoptosis induced by focal brain ischemia reperfusion injury and protect the nerve cell to a certain extent.
结论DG可明显抑制局灶性脑缺血-再灌注所诱导的脑神经细胞凋亡，起到一定的神经保护作用。
Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.
结论：老龄大鼠脑缺血再灌注脑组织损伤与钙超载和自由基损伤有关。
Conclusion the brain tissue extract of rats after acupuncture preconditioning has the effect to resist the cerebral ischemia-reperfusion injury.
结论：针刺预处理脑组织提取液具有明显的抗脑缺血再灌注损伤作用。
Conclusion Inhalation of 0.9% or 1.5% isoflurane can protect brain from focal ischemia reperfusion injury to some extent in rats.
结论缺血前吸入0.9%和1.5%异氟醚对大鼠局灶性脑缺血，再灌注损伤，可产生一定程度的保护作用。
Purpose to investigate the anti-inflammatory effect of angiotensin-converting enzyme inhibitor captopril on the brain against ischemia-reperfusion injury and explore its mechanism of action.
目的观察血管紧张素转化酶抑制药卡托普利对大鼠局灶性脑缺血再灌注损伤后炎症反应的影响。
Objective To study the effects of Naoxing Nasal Spray(NNS)on ultrastructure of brain tissues in rats with focal cerebral ischemia -reperfusion injury(CIRI ).
目的研究脑醒喷鼻剂对大鼠局灶性脑缺血再灌注损伤脑组织超微结构的影响。
Objective:To study the change of the content of transmitter amino acids of cerebral cortex and hippocampus in mice brain after repetitive ischemia-reperfusion(I/R)injury.
提示脑缺血性损伤与脑组织内递质性氨基酸兴奋抑制失衡有密切关系。
Conclusions the brain ischemia reperfusion injury in young and the aged rats were correlated with the decrease of NO level and the increase of TNF.
结论青年大鼠和老龄大鼠脑缺血再灌注脑组织损伤与NO含量降低和TNF增高有关。
Conclusion: Qileng Decoction can protect the brain cerebral ischemia reperfusion injury.
结论芪棱汤对大鼠局灶性脑缺血-再灌注损伤有保护作用。
Expression of AQP4 in the brain at the early period of cerebral ischemia reperfusion injury was increased after injection of APQ4 gene into the rat brain.
脑内注射AQP4基因后可升高大鼠脑缺血再灌注损伤早期脑内aqp4的表达水平。
Objective To explore the effect of Ginkgo biloba extract on the expression of heat shock protein 70(HSP70) in brain tissue of rats with ischemia-reperfusion injury.
目的探讨银杏叶提取物对大鼠缺血再灌注损伤热休克蛋白70表达的影响。
Objective: To study the mechanism of protective function of hirudo injection on ischemia-reperfusion injury of brain in rats.
目的：探讨水蛭注射液对缺血再灌注大鼠脑的保护机制。
Objective To investigate the protective effects of propofol on the brain in ischemia-reperfusion (I/R) injury rats and the underlying mechanism.
目的探讨丙泊酚预处理对大鼠全脑缺血再灌注损伤的保护作用。
CONCLUSION It indicated that tea catechins had protective effects on the injury of cerebral ischemia and reperfusion in rats, and these effects may be related to calcium concentrations in brain.
结论茶儿茶素可能通过降低脑细胞内钙离子浓度，保护脑组织对抗缺血再灌注损伤。
In our study we investigated the influence of ginseng total saponin on brain tissue injury after ischemia-reperfusion by the model of brain ischemia-reperfusion with rat models.
本课题利用大鼠脑缺血再灌注模型，观察人参总皂甙对缺血再灌注后脑组织损伤的影响，目的是探讨人参总皂甙对大鼠缺血再灌注脑组织的保护作用机制。
CONCLUSION: MT shows significantly protective effect against repeated ischemia reperfusion injury in mice, and the mechanism might be related to inhibit the rise of MDA and NO in brain tissue.
结论：MT对反复脑缺血再灌注损伤有显的保护作用，其机制可能与抑制脑水肿及抑制脑内升高的MDA、NO有关。
AIM: To investigate the effects of rolipram on the ability of learning and memory and the activity of PDE4 in hippocampus following the focal brain injury induced by ischemia - reperfusion in rats.
目的：观察咯利普兰对局灶性脑缺血-再灌注损伤大鼠学习记忆能力及海马pde4活性的影响。
Hyperglycemia significantly increases brain damage, mortality rate, and long-term disability after cerebral ischemia and reperfusion injury.
高血糖症会显著增加脑损伤，导致病死率升高，并由脑缺血再灌注损伤后导致患者长期残疾。
Result: PNS could reduce infarct volume of cerebral ischemia-reperfusion injury in rats, reduce the extent of the damage in blood-brain barrier significantly.
结果：三七总皂苷可有效降低脑缺血再灌注损伤大鼠的脑梗死体积，明显减轻脑缺血区血脑屏障破坏的程度。
Result: PNS could reduce infarct volume of cerebral ischemia-reperfusion injury in rats, reduce the extent of the damage in blood-brain barrier significantly.
结果：三七总皂苷可有效降低脑缺血再灌注损伤大鼠的脑梗死体积，明显减轻脑缺血区血脑屏障破坏的程度。
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