Conclusion Carbon tetrachloride has genetic toxic effect on the PCE cell and germ cell in the mice.
结论四氯化碳对小鼠生殖细胞和血红细胞有遗传毒性作用。
Carbon tetrachloride and toluene appear to be the optimum solvents.
四氯化碳和甲苯为较好的溶剂介质。
Under the combination effects of ultrasonic and laser, the changes of scatter spectrum of carbon tetrachloride and glycol samples are studied, and the results of the experiments are discussed.
研究了在超声与激光的联合作用下四氯化碳和乙二醇样品的散射光谱的变化，并对实验结果进行了讨论。
The hydrogen peroxide catalytic oxidation and flocculation treatment technology of wastewater with chloroform, carbon tetrachloride in a methane chloride production process were introduced.
介绍了甲烷氯化物生产过程中采用过氧化氢催化氧化-絮凝法处理含氯仿、四氯化碳废水的技术。
Methods: Experimental model of hepatic fibrosis was induced by carbon tetrachloride and supplementary high-lipid and low-protein diet and alcohol-containing beverage.
方法：采用四氯化碳诱发大鼠肝损伤，辅以高脂、低蛋白复合饲料和一定浓度乙醇饮料，复制肝纤维化模型。
Carbon tetrachloride(CTC) as an ozone depleting substance(ODS) and a feedstock of Freon series compounds has been phased out.
四氯化碳作为氟里昂的生产原料和一种破坏臭氧层物质面临淘汰。
Objective To explore the curative effect of newborn bovine liver bioactive peptide on chemical liver injury of mice caused by carbon tetrachloride.
目的观察新生牛肝活性肽对四氯化碳引起的小鼠化学性肝损伤的保护作用。
Objective To study the genetic toxicity of carbon tetrachloride for male mice and assess the potential genetic toxic effect on mankind.
目的检测环境污染物四氯化碳的诱变活性，评价其可能的潜在遗传毒性。
Carbon tetrachloride is a chemical destroying the ozone layer, and its use should be decreased gradually.
四氯化碳是一种能破坏臭氧层的化学物质，应该逐步减少其使用量。
The research is made on dewaxing shellac by centrifugal subside and refining shellac-wax by Carbon tetrachloride extraction.
介绍了用离心沉降对紫胶脱蜡和四氯化碳萃取精制紫胶蜡的研究过程和结果。
Methods Experimental liver fibrosis rats models of were induced by high fat diet, low fat diet plus ethanol, high fat diet plus ethanol, and carbon tetrachloride subcutaneous injection, respectively.
方法分别建立高脂饮食、低脂酒精饮食、高脂酒精饮食和四氯化碳大鼠实验性脂肪肝肝纤维化模型，用免疫组织化学方法观察造模肝组织中弹性蛋白的表达。
Therefore, it is important for the catalytically hydro dechlorination of carbon tetrachloride to chloroform.
因此，通过四氯化碳催化氢化脱氯生成氯仿显得尤为重要。
The production and use of tetrachloride carbon do great harm to human beings.
四氯化碳的生产和使用，给人类带来了较大危害。
And wipe method with defatted, solution (carbon tetrachloride), environmental pollution and the waste of human body body harmful.
而用擦拭法脱脂，溶液(四氯化碳)浪费大、污染环境且对人身体有害。
If not, anhydrous alcohol and carbon tetrachloride are feasible.
如果没有，无水酒精和四氯化碳都是可行的。
ObjectiveTo study the preventive and therapeutic effects of Concentrated Yigan Decoction on liver fibrosis in rats induced by carbon tetrachloride (CCl 4).
目的研究“益肝浓缩煎剂”对四氯化碳大鼠肝纤维化预防与治疗作用。
The radical 2 as an electron donor underwent a rapid photo-induced redox reaction in carbon tetrachloride leading to the formation of the corresponding piperidine hydroxylamine hydrochloride.
自由基2作为电子给体在四氯化碳中可发生光诱导的氧化还原反应，导致生成相应的哌啶羟胺盐酸盐；
Acetone, tetrahydrofuran, butyl acetate and carbon tetrachloride in liquid state were used to induce the crystallization of bisphenol-A polycarbonate (BAPC) film.
采用丙酮、四氢呋喃、乙酸丁酯和四氯化碳等有机溶剂对双酚a聚碳酸酯(BAPC)进行诱导结晶。
Objective To establish the mice acute liver injury model induced by carbon tetrachloride and to apply in health food screening.
目的通过建立小鼠急性肝损伤模型，并将该模型应用于保健食品保护化学性肝损伤功能的筛选。
In the reaction of chlorosulfonation carbon tetrachloride was used as solvent, adding the sodium chloride in later stage reaction.
在氯磺化反应中引入了四氯化碳溶剂，在反应后期加入氯化钠。
Objective To develope a carbon tetrachloride-induced cirrhotic model in rabbit.
目的应用四氯化碳造成兔肝硬化模型。
Methods:The mice fibrosis liver model was set up by hypodermic injection of carbon tetrachloride.
方法采用皮下注射四氯化碳的方法制造肝纤维化模型，检测并分析血清IL-8的水平。
Using 3, 5-dimethyl phenol as raw materials, sulfuryl chloride as chlorination reagent and carbon tetrachloride as solvent to synthesize chloroxylenol.
以3,5 -二甲酚为原料，硫酰氯为氯化剂、四氯化碳为溶剂，合成对氯间二甲酚。
A chemical process in which methane is chlorinated to yield carbon tetrachloride and numerous other chlorinated hydrocarbons.
甲烷氯化生成四氯化碳和许多其他氯化烃类的化学过程。
CONCLUSION: CSBTA has obviously protective effects on acute liver injury induced by carbon tetrachloride, and can block the formation of rat hepatic fibrosis produced by carbon tetrachloride.
结论：岩黄连总碱对四氯化碳所致急性肝损伤具有明显保护作用，也可抑制大鼠慢性肝纤维化的形成。
Objective To investigate the protective effects of Forsythoside (FOS) on carbon tetrachloride (CCl 4) -induced liver damage in mice.
目的探讨连翘酯苷对四氯化碳肝毒性的保护作用。
Methanol, n-propyl alcohol, acetonitrile and carbon tetrachloride were selected as "molecular probe".
选用甲醇、正丙醇、乙腈、四氯化碳作探针分子。
Results 12 weeks after carbon tetrachloride administration, the rabbit liver developed typical pseudo-lobule, and the liver hydroxyproline content increased 3.5 times compared to normal control.
结果四氯化碳注射12周后，兔肝组织出现典型的假小叶，肝组织羟脯氨酸含量较正常组升高3.5倍。
Those "background" toxics, including carbon tetrachloride, account for the remaining 45 percent of overall cancer risk.
这些“背景”毒物包括四氯化碳，占整体罹患癌症的风险百分之45。
Conclusions: SOD extract of wild Cili has some preventive and therapeutic effect on chronic liver damage induced by carbon tetrachloride.
结论：口服植物源性野生刺梨SOD制剂作为抗氧化剂对大鼠慢性四氯化碳肝损伤有一定预防保护和治疗作用。
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