Objective To investigate the expression of heat shock protein 27 (HSP27) in human age related cataract lens epithelial cells.
目的探讨热休克蛋白(HSP)27在老年性白内障晶状体上皮细胞中的表达及意义。
ResultsBy DNA fragmentation assay, the DNA "ladder" were found in senile cataract lens epithelial cells, while no DNA "ladder" were found in normal cells;
结果DNA片段分析显示,老年性白内障患者晶状体上皮细胞有DNA“梯状”图谱,而健康成年人晶状体上皮细胞未见DNA 断裂现象;
But till up now it was rarely known for the mechanism of contusion cataract, so was the accurate report for the ultrastructure of lens epithelial cells.
但是迄今硕士研究生学位论文为止,对钝挫伤白内障发病机制的研究尚不多见,也未见到其晶状体上皮细胞凋亡超微结构的明确报道。
AIM: To study the effect of heat shock protein-70 (HSP-70) on lens epithelial cells (LECs) in human diabetic cataract.
目的:为探讨热休克蛋白- 70对人类糖尿病性白内障晶状体上皮细胞的影响。
Conclusions HPM may cause lens injury and cataract formation. HPM-induced proliferation disorder in lens epithelial cells may possesses a critical role in cataract development.
结论H PM可导致晶状体损伤及白内障发生,辐射引起上皮细胞增殖异常是白内障发生的重要原因。
Expression of Matrix Metalloproteinase-2, -9 and Their Inhibitors TIMP-1, -2 on Lens Epithelial Cells of Aged-related Cataract.
基质金属蛋白酶-2,9及其抑制因子- 1,2在老年性白内障晶状体上皮细胞的表达。
Objective: To investigate the DNA damage in the lens epithelial cells in cataract patients.
目的:欲研究白内障病人晶状体上皮细胞是否存在DNA损伤及损伤的程度。
Objective: To investigate the DNA damage in the lens epithelial cells in cataract patients.
目的:欲研究白内障病人晶状体上皮细胞是否存在DNA损伤及损伤的程度。
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