Their work indicates that the expression of specific cell surface molecules on tumour cells correlates with clinical parameters.
他们的工作提示肿瘤细胞表面的特殊分子的表达与临床参数有关。
To induce tumor cell apoptosis effectively, we should select tumor cell- specific gene and inhibit its expression specifically.
选择肿瘤细胞特异表达基因并特异性地抑制其表达，是靶向诱导肿瘤细胞凋亡的关键。
A novel method was designed for disease specific B cell epitope mapping and epitope expression in e.
设计了一种新的病原体蛋白质B细胞抗原表位的筛选和重组表达方法。
HDAC and histone deacetylase inhibitors (HDI) can affect expression of some specific genes and induce cell differentiation growth arrest and apoptosis through regulating histone acetylation level.
组蛋白脱乙酰基酶与组蛋白脱乙酰基酶抑制剂通过对组蛋白乙酰化水平的调控，影响特定基因的表达，改变细胞周期的进程。
Methods the expression rates of endothelial cell surface adhesion molecules VCAM-1 and CD62E were detected by flow cytometry (FCM) and specific labeled monoclonal antibody.
方法采用流式细胞术和特异性标记单克隆抗体，测定内皮细胞表面黏附分子VCAM - 1及CD62E的表达率。
Conclusion: Recombinant expression vector PGL3-DF3-DTA could produce specific lethal effect on human breast cancer cell line of DF3 positive.
结论：重组表达载体PGL3 - DF 3 - DTA能对DF 3阳性的乳腺癌细胞产生特异性杀伤作用。
Conclusion the recombinant expression vector PGL3-DF3-DTA can produce a specific killing effect on the DF3-positive human breast cancer cell line.
结论重组表达载体PGL3 -DF 3 - DTA能对DF 3阳性的乳腺癌细胞产生特异性杀伤作用。
Here, we demonstrate that doxorubicin, 5-fluorouracil, and UV and ionizing radiation elicit changes in TLR expression that are cell line - and damage-specific.
研究证实，阿霉素、5氟尿嘧啶、紫外线以及电离辐射均可以细胞系和损伤特异性的方式引起TLR表达的改变。
Here, we demonstrate that doxorubicin, 5-fluorouracil, and UV and ionizing radiation elicit changes in TLR expression that are cell line - and damage-specific.
研究证实，阿霉素、5氟尿嘧啶、紫外线以及电离辐射均可以细胞系和损伤特异性的方式引起TLR表达的改变。
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