This research focused on cell cycle checkpoints and apoptosis induced by DNAdamage in mammalian cells.
本文研究了DNA损伤在哺乳动物细胞中激活细胞周期检查点和引起细胞凋亡的机制。
Conclusion HCMV induces elevated levels of P53, which may affect its normal function in regulating checkpoints of cell cycle, and facilitate its own replication in host cell.
结论HCMV的感染改变了细胞内P 53的水平，可能影响其在细胞周期中的正常功能，营造有利于病毒复制的环境。
However, further studies have indicated that they are actually DNA-damage-response kinases, regulating more than just cell-cycle checkpoints.
进一步的研究表明它们事实上是DNA损伤-应答激酶，不仅仅是调控细胞周期的检查点。
However, further studies have indicated that they are actually DNA-damage-response kinases, regulating more than just cell-cycle checkpoints.
进一步的研究表明它们事实上是DNA损伤-应答激酶，不仅仅是调控细胞周期的检查点。
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