OBJECTIVE: To study the protective effects of TF on cerebral hypoxia in mice.
目的：研究TF对小鼠脑缺氧的保护作用。
AIM To study the protective effects of the cerebrolysin against cerebral hypoxia injury in mice and fetal rats.
目的：观察脑活素对小鼠及大鼠胚脑组织细胞缺氧时的保护作用。
The animal models of cerebral hypoxia-ischemia were prepared in 7 days old rats by systemic hypoxia for 2h and right common carotid artery ligation.
结扎并低氧处理7日龄大鼠右侧颈总动脉制备半球性脑缺血损伤模型。
Conclusions When the brain is severely injured, there is cerebral hypoxia or ischemia during the first 24 hours, and cerebral hyperemia in the following second-fourth days.
结论重型脑损伤后24小时内存在脑缺氧、缺血，伤后第2 - 4天为脑氧合过度、脑充血。
The changes in cerebral hemodynamics tightly connect with the degree of cerebral injuries after hypoxia.
在脑缺氧损伤时，脑血流参数的改变与损伤程度密切相关。
Objective: To study the fetal cerebral blood flow hemodynamics in predicting the diagnostic value of intrauterine hypoxia.
目的：研究胎儿脑血液动力学指标，对预测宫内缺氧的临床价值。
BACKGROUND: brain injury often causes secondary cerebral ischemia and hypoxia, which aggravate the brain damages.
背景：脑损伤后常引起继发性脑缺血、缺氧，使脑损害加重。
AIM: To analyze the influence of high-altitude hypoxia on the expression of cerebral cortex, hippocampus neurogranin in rats in relation to learning and memory abilities.
目的：分析高原低氧环境对大鼠大脑皮质、海马神经颗粒素表达的影响以及与学习记忆的关系。
Results Non-uniform-permanent magnetic field can reduce deformation in neurons of cerebral cortex, roughness of membrane structure, organelle decreasing and etc, which were all caused by hypoxia.
结果不均匀恒磁场可缓解因缺氧而导致的小鼠大脑皮质神经元胞核变形、核膜不光滑及细胞器数量减少等现象。
Objective Observing neuroglobin expression of cerebral cortex neuron in the course of acute repeated hypoxia mice to offer hypoxic endurance foundation.
目的探讨小鼠在急性重复低氧过程中脑皮质神经元的脑红蛋白表达变化。
Objective:To study the protective effect of leech extract on primary cultured neonate rat cerebral cortical neurons from the apoptosis caused by hypoxia in vitro.
目的：研究水蛭提取物对体外缺氧性新生大鼠大脑皮层神经细胞凋亡的保护作用。
Objective To study the effects of Nao Yi An(NYA) serum on the expression of vascular endothelial growth factor(VEGF) gene in cultured rat cerebral microvascular endothelial cell(BMEC)with hypoxia.
目的观察脑溢安血清对体外缺氧培养的大鼠脑微血管内皮细胞血管内皮生长因子（VEGF）基因表达的影响。
Aim: to establish a hypoxia model of cerebral neuron culture and supply a reliable method to the research of the prevention of hypoxia.
目的：建立皮层神经元缺氧损伤的模型，为抗缺氧研究提供可靠的实验方法。
AIM: to investigate the mechanism by which low molecular weight heparin (LMWH) inhibits apoptosis of primary cultured cerebral cortical neurons caused by hypoxia.
目的：探讨低分子肝素对体外培养的新生大鼠大脑皮层神经细胞缺氧性凋亡的作用机制。
Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
Objective: Explore the effects of YTG on red cells deformation ability of rat with cerebral ischemia and mouse's decompression hypoxia ability.
目的：探讨养阴通脑颗粒对脑缺血大鼠红细胞变形能力和小鼠减压缺氧耐力影响。
Results Many factors and diseases were related to BGC, such as hypothyroidism, intrauterine infection, intrauterine hypoxia, epilepsy, posttraumatic cerebral lacunar infarction.
结果多种因素、疾病与儿童BGC相关，如甲状旁腺功能减退、宫内感染和缺氧、癫、外伤后腔隙性脑梗死等。
This study suggests that long term residing at high altitude results in an increase in cerebral vasodilative response and a decrease in pulmonary vasoconstrictive response to acute hypoxia.
结果提示：高原移居平原者与世居平原者相比，脑血管缺氧性扩张反应较强，而肺血管缺氧性收缩反应较弱。
This article introduces the effect of high central nervous system, changes of cerebral blood flow after hypoxia, damage of hypoxia to neurons in the respiratory center, neurotransmitters a…
本文主要介绍高级中枢、缺氧后脑血流量变化、缺氧对呼吸中枢神经元的实质性损害、 神经递质和神经调质在缺氧性呼吸抑制过程中的作用。
Conclusions Opening ETS can cause cerebral ischemia and hypoxia during intracranial hypertension.
结论颅内高压状态下进行开放式ETS可引起脑缺血、缺氧反应。
Objective to investigate the effect of hypoxia on cerebral intracellular biological oxidation and to find out the effective ways of drug intervention.
目的了解缺氧对新生鼠脑细胞生物氧化的影响，寻找有效的干预治疗方法。
RESULTS The compound loach powder increased the ability of rats'bearing hypoxia, survival time of cerebral circulation disturbance and the weight of immune organs.
复方泥鳅粉延长小鼠耐缺氧、脑循环障碍及存活时间，提高免疫器官质量。
Conclusion It is suggested that, the increase of ET and NO contents of cerebral cortex and thalamus may participate in the pathogenesis of hypoxia brain injury in old rats.
结论急性低压缺氧可引起老龄大鼠大脑皮质和丘脑中et、NO含量明显升高以及大脑皮质血管内皮细胞和神经细胞明显损伤，ET、NO可能参与了缺氧性脑损害的病理过程。
Objective Observing the ultrastructure change of cerebral cortex neuron in the course of acute repeated hypoxia mice to offer morphological foundation.
目的观察小鼠在急性重复低氧过程中脑皮质神经元的超微结构变化。
In neonatal stadium, 1 patient was found with hypoxia-ischemic encephalopathy, and 4 with cerebral hemorrhage.
新生儿期发现缺血缺氧性脑病1例，颅内出血4例。
Through the hypoxia tolerance test in ordinary pressure and swimming test taking mice as object, the mechanism of improving cerebral oxygen supply and toxicology of SHENQI BUCCAL. TABLET was studied.
通过小鼠常压耐缺氧实验、小鼠游泳实验探讨了参七含片改善脑供氧作用机理，并对其急性毒性和长期毒性进行了初步考察。
Through the hypoxia tolerance test in ordinary pressure and swimming test taking mice as object, the mechanism of improving cerebral oxygen supply and toxicology of SHENQI BUCCAL.
通过小鼠常压耐缺氧实验、小鼠游泳实验探讨了参七含片改善脑供氧作用机理，并对其急性毒性和长期毒性进行了初步考察。
However, in hypoxia and hypercapnia, there are many controversies about the regulation of nitric oxide on cerebral blood vessels. In this paper, we have reviewed the latest advances in this field.
但关于它在低氧和高二氧化碳条件下对脑血管是否具有调节作用还存在着争议。
Preterm newborn child suffering from epilepsy in a higher proportion of perinatal asphyxia, chronic functional brain hypoxia, congenital dysplasia, and so is the cause of cerebral ischemia.
新生儿中早产儿患上癫痫病的比例较高，围产期窒息、慢性功能性脑缺氧、先天性脑发育不良、脑缺血等是致病原因。
Preterm newborn child suffering from epilepsy in a higher proportion of perinatal asphyxia, chronic functional brain hypoxia, congenital dysplasia, and so is the cause of cerebral ischemia.
新生儿中早产儿患上癫痫病的比例较高，围产期窒息、慢性功能性脑缺氧、先天性脑发育不良、脑缺血等是致病原因。
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