CONCLUSION Hyperoxia solution can mitigate the cerebral ischemia and reperfusion injury.
结论高氧液对神经元缺血损伤具有保护作用。
Conclusion Mild hypothermia therapy may inhibit the expression of HSP70 and GFAP after cerebral ischemia and reperfusion injury in rats.
结论亚低温能减轻大鼠脑缺血再灌注损伤，降低脑组织HSP70及GFAP蛋白的表达。
Hyperglycemia significantly increases brain damage, mortality rate, and long-term disability after cerebral ischemia and reperfusion injury.
高血糖症会显著增加脑损伤，导致病死率升高，并由脑缺血再灌注损伤后导致患者长期残疾。
Methods: the cerebral ischemia and reperfusion inJury model of rabbits was established to observe the protective effect of Naomaitong on them.
方法：通过兔全脑缺血-再灌注的病理模型，观察中药脑脉通对其损伤的保护作用。
The protective effect of PTFK granule on cerebral ischemia and reperfusion injury might be relation to regulating balances of ET, CGRP, SOD and NO in plasma or serum.
偏瘫复康颗粒对脑缺血再灌注损伤所具有的保护作用可能和参与调节et、CGRP、NO,SOD等因素有关。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Objective To investigate the effects of high thoracic epidural anesthesia (HTEA) on cerebral ischemia-reperfusion (I/R) injury, and the possible mechanism.
目的探讨上胸段硬膜外阻滞（HTEA）对大鼠脑缺血再灌注损伤的作用及可能机制。
Objective To study the effects and mechanism of Naotaifang extract preconditioning on the cerebral ischemia-reperfusion injury in gerbil.
目的：研究脑泰方提取物预处理减轻沙土鼠脑缺血再灌注损伤的作用及作用机理。
Objective To explore the protective effect of baicalin on the cerebral ischemia-reperfusion injury in rats and its mechanism.
目的观察黄芩苷对大鼠脑缺血再灌注损伤的保护作用，并初步探讨其作用机制。
OBJECTIVE: to study the influence of different doses of naloxone on focal cerebral ischemia reperfusion injury in rats and to explore its mechanism.
目的：观察不同剂量的纳洛酮对大鼠局灶性脑缺血再灌注损伤模型的影响，并探讨其保护作用机制。
Objective: To observe the expression of poly (ADP-ribose) polymerase (PARP) in focal cerebral ischemia with reperfusion injury and explore the role of PARP in apoptosis.
目的观察多聚ADP 核糖聚合酶（PARP）在脑缺血再灌流损伤中的表达，探讨PARP在细胞凋亡中的作用。
Conclusions Complement and inflammatory reaction participated in cerebral ischemia-reperfusion injury.
结论补体及炎症反应参与脑组织缺血再灌注损伤。
Objective To study the changes of neutrophil chemotactic index and serum IL-8 contents after cerebral ischemia reperfusion injury in rats.
目的：探讨了血清il - 8和中性粒细胞趋化指数测定在脑缺血再灌注损伤中的变化。
Conclusion in the process of focal cerebral ischemia and reperfusion, regulating the quantity of NO in the propriety range may protect and avoid purpose of neuronal cell injury.
结论局灶性脑缺血后，调节NO生成量在适当范围，可以达到保护、防止神经细胞进一步损伤的目的。
BACKGROUND: Cell apoptosis and expression of related apoptotic gene are found in rats with acute cerebral ischemia-reperfusion injury.
背景：大鼠急性脑缺血再灌注损伤后有细胞凋亡及凋亡相关基因的表达。
AIM: To explore the role of endothelin (et) 3 and astrocytes in cerebral ischemia reperfusion injury in diabetic mice.
目的：探讨内皮素-3和星形胶质细胞在糖尿病小鼠脑缺血再灌注损伤中的作用机制。
Objective: To investigate the influence of NGF on neural functional recovery in focal cerebral ischemia reperfusion injury in rabbits and evaluate with MR imaging.
目的：研究神经生长因子(NGF)对兔局灶性脑缺血再灌注损伤神经功能修复的影响，并利用MR成像技术进行评价。
Methods: adopt reversible cerebral ischemia reperfusion injury model in rats, acupuncture the points - "Renzhong", "Zhongchong" and "Fengfu", calculate the content of CK and LDH using testing box.
方法：采用可逆性脑缺血再灌注损伤大鼠模型，通过电针“人中”穴、双侧“中冲”穴及“风府”穴，利用CK及LDH测试盒测试酶活性。
Objective To measure the protective effects of Naogengyin on cerebral ischemia-reperfusion injury in rat and its mechanisms.
目的观察脑梗饮对大鼠局灶性脑缺血再灌注脑组织损伤的保护作用，并进一步探讨其作用机制。
To study the therapeutic window of oppourtunity for VEGF following focal cerebral ischemia reperfusion injury in rabbits and the effectiveness was evaluated by advanced MR imaging technology.
目的：探讨MR影像学评价血管内皮细胞生长因子对家兔局灶性脑缺血再灌注损伤的有效治疗时间窗。
CONCLUSION: the illegibility of DNA and RNA fluorescent response in operation group is related with the breakage of DNA chain induced by oxidative stress during cerebral ischemia-reperfusion injury.
结论：手术组DNA和RNA荧光反射不清，可能与脑缺血再灌流损伤中氧化应激引起DNA链断裂有关。
CONCLUSION: Diabetes is one of the important causes of cerebral ischemia reperfusion injury, and et 3 and activated astrocytes may worsen the neural injuries in diabetic mice.
结论：糖尿病是脑缺血再灌注损伤重要因素之一；内皮素-3和星形胶质细胞激活可能是糖尿病小鼠神经细胞损伤加重恶化的机制之一。
CONCLUSION: Propofol and midazolam, but not thiopental sodium, can provide protective effects against reperfusion induced injury in rats subjected to focal cerebral ischemia.
结论：丙泊酚和咪达唑仑有拮抗大鼠局灶脑缺血后再灌注损伤的作用。
Objective It is to explore the treating and preventing effect of Ligustrazine on cerebral ischemia reperfusion injury.
目的探讨川芎嗪注射液对脑缺血再灌注损伤的防治作用。
Objective:To study the change of the content of transmitter amino acids of cerebral cortex and hippocampus in mice brain after repetitive ischemia-reperfusion(I/R)injury.
提示脑缺血性损伤与脑组织内递质性氨基酸兴奋抑制失衡有密切关系。
Objective to explore the expressions of growth associated protein-43 (GAP-43) and insulin-like growth factor-1 (IGF-1) in nervous system after cerebral ischemia reperfusion injury in rats.
目的探讨大鼠脑缺血再灌注损伤后生长相关蛋白- 43 (GAP - 43)和胰岛素样生长因子- 1 (IGF - 1)的表达。
METHODS:The models of cerebral ischemia reperfusion injury and senile dementia mice caused by D galactose were used.
方法：采用脑缺血再灌注损伤及D 半乳糖所致痴呆小鼠模型。
Objective To investigate the effects of simvastatin on cerebral injury induced by ischemia reperfusion in rats and the mechanisms.
目的探讨辛伐他汀抗大鼠脑缺血再灌注损伤的作用及机制。
Objective:To investigate the influence of Radix Salviae Miltiorrhiae(RSM) on adhesion between leukocyte and endothelial cell in the focus of cerebral ischemia-reperfusion injury.
前言： 目的：研究丹参对脑缺血再灌注损伤后局灶区白细胞与内皮细胞粘附性变化及影响；
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