ObjectiveTo investigate the mechanism of cerebral white matter damage in premature rats induced by intrauterine infection.
目的探讨宫内感染致早产鼠脑白质损害的机制。
Objective: To evaluate the usefulness of diffusion tensor tracking in abnormal cerebral white matter induced by cerebral gliomas.
目的：评价磁共振弥散张量纤维束成像技术在大脑胶质瘤所导致的大脑白质纤维异常改变中的价值。
ConclusionIt is similar to single preterm, cerebral white matter injury is the major forms of MRI in twin preterm with brain injury.
结论与单胎早产儿相似，脑白质损伤也是双胎早产儿脑损伤的MRI的主要表现形式。
Logistic regression analysis revealed that LDL and hypertension were independent risk factors for cerebral white matter chronic ischemia.
逻辑回归分析提示LDL和高血压是脑白质慢性缺血的单独致病危险因素。
Minor features include dental pits, renal cysts, rectal hamartoma polyps, cerebral white matter migration lines, gingival fibromas, and confetti skin lesions.
辅助特征包括牙齿凹痕、肾囊肿、直肠错构瘤样息肉、脑白质放射移行线，牙龈纤维瘤，斑驳样的皮肤斑。
The abnormal signals of neuroradiologic imaging predominantly showed symmetrical lesions in the posterior cerebral white matter bilaterally, and also involved the cortex.
神经影像学以双侧对称性大脑后部白质为主的异常信号，也可累及皮层。
The presence of hemorrhage in patchy cerebral white matter lesions instead suggests the diagnosis of Acute Hemorrhagic Encephalomyelitis, which is distinct from ANE and ADEM.
在斑片状的白质病变内出血出血提示急性出血性脑脊髓炎的诊断，这可以与ADEM和ANE相鉴别。
Objective to examine the relationship between cerebral white matter lesions (WML) of different severity and the cognitive impairment in old people with vascular risk factors.
目的探讨存在血管危险因素老年人不同程度脑白质损害(WML)与认知障碍的关系。
Lacunar infarcts were rated visually. Volumes of deep white matter and WML and presence of lacunar infarcts reflected cerebral small vessel disease.
人工视觉判断腔隙性梗塞的程度，深灰质的容积、脑室容积及腔隙性梗塞的存在与否反应脑部小血管病变的程度。
Involvement of adjacent white matter tracts, cerebral cortex and cerebellar structures has also been described but is less common.
邻近白质束、大脑皮质及小脑结构的受累也可以出现，但是相对不常见些。
The lesion is well demarcated (Figures 1 ~ 4 in the first batch) and found in the white matter only. The cerebral cortex is spared (Figure 3).
这种疾病境界清楚(第一组图1 -4)，而且只发生于大脑白质，皮质未累及(图3)。
PML lesions are found in white matter and at the corticomedullary junction of cerebral and cerebellar cortex.
PM L病变见于脑和小脑的白质和皮质-白质交界处。
Damage to the white matter beneath the cerebral cortex.
对大脑皮层之下的脑白质的损毁。
Fractional anisotropy (FA) were measured in some main white matter structures of peripheral white matter, basal ganglia, cerebral peduncle etc.
分别在半卵圆中心、基底节区和大脑脚层面测量主要白质束的FA值。
MR imaging showed multiple, scattered, FLAIR hyperintense foci in bilateral cerebral hemispheres involving deep white matter, right middle cerebellar peduncle, and pons.
MR影像显示双侧大脑半球多发、散在的病变，Flair序列上高信号，主要累及深部白质、右侧小脑中脚、脑桥。
Cerebral white and gray matter move to inside.
脑灰白质向内移位。
There were obvious pathological change in the brain after the chronic cerebral hypoperfusion, such as myelinic degeneration and formation of glial nodule in white matter.
缺血后脑的病理改变以神经细胞变性、固缩和退变，皮层下白质的神经纤维和髓鞘溃变为主，普遍存在胶质细胞增生，形成胶质小结。
The rate of cerebral infarction associated with white matter lesion(WML)and brain atrophy in VD group was higher than that of NVD group. Conclusion :The factors that mosts…
结论：血管性痴呆与梗塞部位及是否存在脑白质变性和脑萎缩有关。
There might be involvement of adjacent white matter tracts, cerebral or cerebellar cortex but this is less likely.
并且可能累及邻近白质束，大脑及小脑皮层，但是可能性要小一些。
There is mild nonspecific periventricular white matter disease and diffuse cerebral atrophy.
片示轻度的非特异性的室周白质病变和弥漫性的脑萎缩。
T2WI phase: cerebral cortex and white matter can better distinguish the signal above the cortical white matter signal, after the bilateral lateral ventricle angle showed a high signal;
T2WI相：脑皮质与脑白质能较好分辨，皮质信号高于白质信号，双侧侧脑室后角呈高信号；
The ratio of FDG uptake of the lesion to normal white matter in PET and the ratio of cerebral blood perfusion of the lesion to normal white matter also were compared.
PET是病灶FDG摄取与正常脑白质比率进行比较，ASL是病灶大脑血环流量与正常脑白质比率进行比较。
The ratio of FDG uptake of the lesion to normal white matter in PET and the ratio of cerebral blood perfusion of the lesion to normal white matter also were compared.
PET是病灶FDG摄取与正常脑白质比率进行比较，ASL是病灶大脑血环流量与正常脑白质比率进行比较。
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