These results proved that polycythemia may play an important role in pathogenesis of chronic hypoxic pulmonary hypertension and cardiac hypertrophy.
以上结果表明红细胞增多可能在慢性缺氧性肺动脉高压和右室肥大发生起着重要作用。
Conclusion Nitric oxide release decreased during chronic hypoxia, which was probably involved in the mechanism of hypoxic pulmonary hypertension.
结论大鼠慢性缺氧时一氧化氮释放减少，而一氧化氮释放减少可能参与缺氧性肺动脉高压的形成。
Conclusion Nitric oxide release decreased during chronic hypoxia, which was probably involved in the mechanism of hypoxic pulmonary hypertension.
结论大鼠慢性缺氧时一氧化氮释放减少，而一氧化氮释放减少可能参与缺氧性肺动脉高压的形成。
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