Conclusion: the aging rat model induced by D-gal can be used as the model for aging research.
结论：D -半乳糖致衰大鼠可作为研究衰老的模型。
Objective: To observe the ultrastructural changes of arcuate nucleus in hypothalamus of D-gal aging rats.
目的：观察d -半乳糖衰老模型大鼠下丘脑弓状核超微结构的变化。
Methods: The senile mouse models induced by D-gal were prepared and given water extract of Machixian for 30 days.
方法：采用小鼠注射d -半乳糖制成衰老模型。马齿苋水提液灌胃30 d。
Results: The D-Gal-treated rats significantly experienced a dynamic escalation of ALT, AST and IL-18 levels(P<0.05).
结果：D-氨基半乳糖造模后，两组大鼠血清ALT，AST和肝匀浆IL-18值呈动态升高（P<0.05）。
All these imply obvious anti-aging and immune regulation effects of the peptides on the senile mice induced by D-gal.
说明鳙鱼肽对d -半乳糖致衰小鼠有明显的抗衰老和免疫调节作用。
Objective to observe the effect of Jiangzhi Ninggan capsule on acute hepatic injury induced by D-galactosamine (D-Gal) in mice.
目的：观察降脂宁肝胶囊对大鼠脂肪肝的治疗作用并初步探讨其作用机制。
Objective: To investigate the liver morphology and the oxidation and anti-oxidation capability of aging rat model induced by D-gal.
目的：观察d -半乳糖致衰大鼠肝脏形态学改变及氧化水平和抗氧化能力的变化。
METHODS The mice with decrepit model induced by D-galactose (D-gal) were treated with LZS of different doses respectively for 4 weeks.
方法：采用半乳糖腹腔注射制作大鼠衰老模型，分别用不同剂量的雷竹笋汁灌胃4周。
CD137 and CD28 can promote the activation and survival of t cells in aged and D-gal group. But CD28 has stronger effect on regulation of t cells than CD137.
CD 137及CD 28分子对两组衰老T细胞有近似的调节作用，均能促进衰老T细胞的活化及存活，CD 28分子的调节作用强于CD 137分子。
CD137 and CD28 can promote the activation and survival of t cells in aged and D-gal group. But CD28 has stronger effect on regulation of t cells than CD137.
CD 137及CD 28分子对两组衰老T细胞有近似的调节作用，均能促进衰老T细胞的活化及存活，CD 28分子的调节作用强于CD 137分子。
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