D-galactose was used to build aging model.
利用D-半乳糖建立衰老模型。
Methods Subacute senile mice model was established by injection of D-galactose.
方法通过注射D半乳糖建立亚急性衰老小鼠模型。
Methods Subacute senile mouse model was established by injection of D-galactose.
方法通过注射d半乳糖建立亚急性衰老小鼠模型。
Calculate the percentage composition of the two forms of D-galactose at equilibrium.
计算达到平衡时这两种形式的D -半乳糖的百分含量。
To study the antioxidant effect of the hawthorn extract on D-galactose induced aging mice model.
探讨山楂提取物对d -半乳糖致衰小鼠抗氧化系统的影响。
Conclusion: Puerariae can significantly inhibit glycation reaction in rats induced by D-galactose.
结论：葛根素对d -半乳糖诱导的大鼠糖基化反应具有明显的抑制作用。
OBJECTIVE to investigate the influence of canthaxanthin on the D-galactose induced osseous in rats.
目的研究角黄素对d -半乳糖致衰老大鼠骨质的影响。
Methods: Y-electronic maze was used to study the behavior of dementia rats induced by D-galactose and QA.
方法：采用D-半乳糖、喹啉酸致痴呆大鼠模型，使用大鼠Y型电迷宫法，研究药物对大鼠学习记忆的影响；
Objective: to study the effect of Dunhuang Weicifang delaying senility to D-galactose presenile model mice.
目的：研究敦煌韦慈方对d半乳糖所致衰老模型小鼠的延缓衰老作用。
Methods: Senile rats model was established by adopting the method that injected D-galactose subcutaneously in nape.
方法：采用D -半乳糖颈后皮下注射的方法，造成亚急性衰老动物模型。
Peritoneal and retrobulbar injection of D-galactose or DL-phenylalanine can induce cataractous changes in rat lens.
半乳糖或DL苯丙氨酸分别经腹腔及半球后注射，均可诱发大鼠产生白内障。
Objective to observe the changes of morphology and biochemical parameters in rats with D-galactose-induced skin ageing.
目的观察d -半乳糖诱导皮肤衰老大鼠的皮肤形态学改变及与衰老相关的生化指标变化。
Objective: to observe the protective effect of tremella polysaccharides (TP) on mouse aging model induced by D-galactose.
目的：观察银耳多糖对D -半乳糖所致衰老小鼠的作用。
Objective: To investigate anti-aging effect and mechanism of Jian-nao-yi-shou capsule on aged mice induced by D-galactose.
目的：观察健脑益寿胶囊的抗衰老作用，并初步探讨其作用机理。
The aging model of mice made by D-galactose was employed in this experiment to study the antiaging effect of "Changshouling".
本实验采用连续给予D-半乳糖的方法建立小鼠衰老模型，并观察中药“长寿灵”的抗衰老作用。
Conclusion: Erzhi Pill can effectively resist the skin senility of the mouse with subacute senility resulting from D-galactose.
结论：二至丸能有效防止D -半乳糖致衰老小鼠的皮肤衰老。
AIM: to observe the protective effect of extract from maize plumule against lipid peroxidation in drosophila induced by D-galactose.
目的：观察玉米幼芽提取物对d -半乳糖引起果蝇脂质过氧化的拮抗作用。
Objective to investigate the effects of Pingmaitong granules on free radical metabolism of D-galactose-induced subacute aging model.
目的探讨平脉通颗粒对D -半乳糖所致亚急性衰老模型自由基代谢的影响。
METHODS The mice with decrepit model induced by D-galactose (D-gal) were treated with LZS of different doses respectively for 4 weeks.
方法：采用半乳糖腹腔注射制作大鼠衰老模型，分别用不同剂量的雷竹笋汁灌胃4周。
Objective To observe the effect of carboxymethyl chitosan (CMC) on the lifespan of Drosophila melanogaster and D-galactose-induced aging in mice.
目的观察羧甲基壳聚糖（CMC）对黑腹果蝇寿命的影响及其对D-半乳糖致亚急性衰老小鼠的抗衰老作用。
AIM: to investigate the effects of aminoguanidine intervention on lens cell damage induced by D-galactose in rat eyes and its mechanism of action.
目的：探讨d -半乳糖致大鼠眼晶体损伤及氨基胍的干预作用及其机制。
Objective To study the effect of oxygen stress induced by D-galactose unite with hyperlipoidemia on hemorheology and carotid blood perfusion in rats.
目的观察D-半乳糖诱发氧应激及实验性高脂血症对大鼠血液流变特性的影响。
Results Compared with the D-galactose induced mouse model, apigenin could improve the ability of escaping electrical stimulation significantly(P<0.01).
结果腹腔注射芹黄素的小鼠学习能力和记忆保持率均显著优于模型对照组（P<0.01）。
Conclusion It was suggested that Pyracanthy had some antioxidation and might prolong the aging process of mice model induced by overdose of D-galactose.
结论火棘有一定的抗氧化作用，并能延缓由D -半乳糖所致衰老小鼠的衰老过程。
AIM: to study the effects of puerarin on learning-memory behavior in aging mice induced by D-galactose and to explore in-brain mechanism of its effects.
目的：探讨葛根素对d -半乳糖致衰老小鼠学习记忆的影响及其作用机制。
Conclusion These results support the idea that transplantation of mesenchymal stem showed a protective effect on intestine damage induced by D-galactose.
结论骨髓间充质干细胞能够一定程度上减轻d -半乳糖致衰老大鼠的小肠损伤。
Conclusion the decreased expression of PBR in hippocampus synaptosomes is possibly associated with the spatial learning-memory impairments induced by D-galactose.
结论海马突触体pbr表达量可能与大鼠空间学习和记忆有关。
Conclusion the decreased expression of PBR in hippocampus synaptosomes is possibly associated with the spatial learning-memory impairments induced by D-galactose.
结论海马突触体pbr表达量可能与大鼠空间学习和记忆有关。
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