AIM: To explore the change of delayed rectifier potassium channel (KV) activity in alveolar macrophages (AM) in chronic obstructive pulmonary disease (COPD) rats.
目的：探讨慢性阻塞性肺疾病(COPD)模型大鼠肺泡巨噬细胞延迟整流钾通道(KV)活性变化。
Ion channel activity had the character of voltage-dependence. Some cells, which did not exhibit sodium currents exhibited, delayed rectifier potassium currents.
并且离子通道活动具有明显的电压依赖性，在没有记录到钠电流的细胞记录到延迟整流钾电流。
In long-QT syndrome type 1, mutations occur in the KCNQ1 gene, which encodes the repolarizing potassium channel mediating the delayed rectifier IKs current.
在I型QT延长综合征中，KCNQ1基因发生突变，对介导延迟整流钾通道电流的复极化钾通道进行编码。
In long-QT syndrome type 1, mutations occur in the KCNQ1 gene, which encodes the repolarizing potassium channel mediating the delayed rectifier IKs current.
在I型QT延长综合征中，KCNQ1基因发生突变，对介导延迟整流钾通道电流的复极化钾通道进行编码。
应用推荐