Method Serum PON1 activity was measured by using paraoxon as substrate in 30 case of norm, 52 of simple fatty liver, 41 of steatohepatitis and 31 of fatty liver cirrhosis.
方法以对氧磷为底物测定30例正常人、52单纯性脂肪肝患者、41例脂肪性肝炎患者、31例脂肪性肝硬化患者血清pon1活性。
Fatty liver disease can take many forms that vary in severity, with the most severe being nonalcoholic steatohepatitis (NASH), fibrosis and cirrhosis. NAFLD can lead to liver failure.
脂肪性肝病恶化后会很复杂，最严重的是非酒精性脂肪肝炎、肝纤维化、肝硬化。非酒精性脂肪肝会导致肝功能衰竭。
Fatty liver disease can take many forms that vary in severity, with the most severe being nonalcoholic steatohepatitis (NASH), fibrosis and cirrhosis.
脂肪性肝病恶化后会很复杂，最严重的是非酒精性脂肪肝炎、肝纤维化、肝硬化。
Non-alcoholic fatty liver disease can result in cirrhosis of the liver or liver cancer.
非酒精性脂肪肝病能够导致肝硬化或肝癌。
Nonalcoholic fatty liver disease(NAFLD) is a kind of chronic liver disease, whose spectrum of the disease includes simple hepar adiposum, steatohepatitis(NASH), hepatic fibrosis, and cirrhosis.
非乙醇性脂肪性肝病（NAFLD）是一种常见的慢性肝病，其疾病谱包括单纯性脂肪肝、非乙醇性脂肪性肝炎（NASH）、肝纤维化及肝硬化。
So, that lays the ground for the first mechanism, which is: increasing the risk of developing fatty liver disease which eventually progresses to cirrhosis.
这是第一个可能机制的基础：增加发生脂肪肝的风险最终会进展为肝硬化。
Results: fatty liver and hepatic cirrhosis models were successfully induced.
结果：脂肪肝及肝硬化模型复制成功。
Conclusion: The CT appearances of chronic alcoholic toxic cerebral injuries and liver injuries were cerebral atrophy in different degree, encephalomalacia foci, fatty liver, liver cirrhosis.
结论：慢性酒精中毒性脑病、肝病的CT表现特点是不同程度的脑萎缩、脑软化灶、脂肪肝、肝硬化。
Cusi said nonalcoholic fatty liver disease probably accounts for about half of the people who develop cirrhosis of the liver.
Cusi认为非酒精性脂肪肝可能为约一半肝硬化患者的病因。
The pathogenesis of nonalcoholic steatohepatitis and other fatty liver diseases: a four-step model including the role of lipid release and hepatic venular obstruction in the progression to cirrhosis.
非酒精性脂肪性肝炎和其他脂肪性肝病的发病机制：四步模型，包括肝硬化进展的脂质释放和肝微梗阻的作用。
The liver injury being caused by various harmful factors were manifested as hepatic necrosis, fatty liver, cholestasis, liver fibrosis, liver cirrhosis and liver cancer and so on.
各种有害因素所致的肝损伤可表现为肝坏死、脂肪肝、胆汁郁积、肝纤维化、肝硬化及肝癌等。
Such four cases as ascites due to cirrhosis, portal hypertension; non-alcoholic fatty liver and jaundice were presented for a detailed explanation.
今举肝硬化腹水、门静脉高压症、非酒精性脂肪肝、黄疸四则验案详细说明之。
Method Fatty liver model rats were made by orotic acid douche, cirrhosis were made by injection of thioacetamide(TAA), and control group was made at the same time.
方法采用脂肪乳剂灌胃复制大鼠脂肪肝模型，用硫代乙酰胺腹腔注射诱导肝硬化动物模型，同时设立正常对照组；
Method Fatty liver model rats were made by orotic acid douche, cirrhosis were made by injection of thioacetamide(TAA), and control group was made at the same time.
方法采用脂肪乳剂灌胃复制大鼠脂肪肝模型，用硫代乙酰胺腹腔注射诱导肝硬化动物模型，同时设立正常对照组；
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