The activity of fibrin monomer against IF 53, inhibited by RGD peptide, were analyzed by rad.
目的 ：研究妊高征患者血浆纤维蛋白原单体聚合功能的临床意义。
Objective: This paper was designed to analyse the molecular mechanism of cell spreading induced by fibrin monomer.
目的：探讨纤维蛋白单体引起细胞伸展的机理。
There was a positive correlation between fibrin monomer polymerization function and scores of clinical evaluation in different degree.
脑梗死患者神经功能缺损程度评分与纤维蛋白聚合功能各项参数呈不同程度的正相关。
Methods Prothrombin fragments 1 and 2 (F1+2), fibrinopeptide A (FPA), soluble fibrin monomer complex(SFMC) were examined by enzyme linked immunoassay(ELISA);
方法采用酶联免疫分析法测定凝血酶原片段1+2 （F1+2 ）、纤维蛋白肽A（FPA）、可溶性纤维蛋白单体复合物（SFMC） ；
Conclusion: Plasma Fbg concentration and fibrin monomer polymerize function were decrease, showed that patients were low coagulation state and contributing to one cause of hemorrhage.
结论：失代偿性肝硬化患者因纤维蛋白原浓度下降致使纤维蛋白单体聚合功能降低，从而出现低凝状态，这可能是构成出血倾向的原因之一。
Polymerization of fibrin monomer was inverse ratio with fibrinogen bound sialic acid. It suggests that fibrinogen boud sialic acid levels may be diagnostic index acquired dysfibrinogenaemia.
表明纤维蛋白原结合唾液酸的水平可作为诊断肝硬化获得性异常纤维蛋白血症的理想指标。
RESULTS:Each parameter indicating the monomer polymerization functions of fibrin in plasma was significantly increased in ischemic cerebrovascular diseases patients than healthy individuals(P< 0.01).
结果：缺血性脑血管病患者较健康人纤维蛋白单体聚合功能各项参数均显著增高（P<0.01）；
RESULTS:Each parameter indicating the monomer polymerization functions of fibrin in plasma was significantly increased in ischemic cerebrovascular diseases patients than healthy individuals(P< 0.01).
结果：缺血性脑血管病患者较健康人纤维蛋白单体聚合功能各项参数均显著增高（P<0.01）；
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