Results:The sequence of the cloned GAP-43 was confirmed to be correct by DNA sequencing.
结果：从大鼠少突胶质前体细胞中克隆到序列正确的GAP-43全长编码序列。
Objective To study the expression of GAP-43 in the central nerve system of developing rats.
目的研究神经生长相关蛋白质(GAP 43)在发育大鼠中枢神经系统的表达及其意义。
Conclusion: GAP-43 and ET-1 play important roles in the pathophysiological process of blood brain barrier damage after diffuse brain lesion.
结论GAP - 43及ET - 1在脑损伤后血脑屏障损害的病理生理过程中起重要作用。
GAP-43 can be used as one of the important indexes during the research of nervous development and growth, in addition, the repair of damage.
在中枢神经系统生长发育和损伤后修复过程中GAP - 43起着重要的作用。
Conclusion: GAP-43 plays an important role in the growth of prominence during the early period of differentiation of NSC into mature neurons.
结论：在神经干细胞分化为神经元过程中GAP - 43基因参与引导突起的生长并发挥重要作用。
Objective: To study the effect of rehabilitative training on behavioral recovery and expression of GAP-43 in spinal cord after cerebral infarction rats.
目的：研究康复训练对实验性脑梗塞偏瘫大鼠行为学和脊髓生长相关蛋白（GAP - 43）表达的影响。
ObjectiveTo study the expression of GAP-43 hippocampal formation during brain developmental critical period of both hypothyroid and excessive iodine rats.
目的研究高碘对甲状腺激素以及甲减、高碘对脑发育临界期大鼠海马组织神经生长相关蛋白表达的影响。
Objective: To study the expression of growth associated protein 43 (GAP-43) in the cerebral development of rats with intrauterine growth restriction (IUGR).
目的：探讨生长相关蛋白43 (GAP - 43)在胎儿宫内发育迟缓(IUGR)鼠脑发育过程中的表达情况。
Objective:To investigate the effects of chondroitinase ABC on chondroitin sulfate proteoglycans(CSPGs) and growth associated protein-43(GAP-43)after spinal cord injury in rats.
目的：探讨硫酸软骨素酶ABC对严重的大鼠脊髓损伤蛋白多糖及生长相关蛋白43（GAP-43）基因表达的影响。
Conclusion: microwave could enhance the expression of GAP-43 and promote the regeneration and functional recovery of the injured peripheral nerve after early microwave irradiation.
结论：周围神经损伤后早期应用微波辐射治疗能增加GAP - 43的表达，促进损伤神经再生及功能恢复。
Conclusions: Jinkui Shenqi Pills can restore the injured spiral ganglion cells by increasing the expression of GAP-43 and NGF in the cochlea of gentamicin-induced deafness guinea pigs.
结论：金匮肾气丸可提高庆大霉素致聋豚鼠耳蜗gap - 43、NGF表达，对螺旋神经节细胞进行修复。
Objective to explore the expression of growth associated protein-43 (GAP-43) to the change of plasticity of neurons axon regeneration after cerebral ischemic reperfusion injury in rats.
目的探讨脑缺血再灌注损伤后生长相关蛋白- 43 (GAP - 43)的表达对神经元轴突再生的可塑性变化。
Objective to explore the expressions of growth associated protein-43 (GAP-43) and insulin-like growth factor-1 (IGF-1) in nervous system after cerebral ischemia reperfusion injury in rats.
目的探讨大鼠脑缺血再灌注损伤后生长相关蛋白- 43 (GAP - 43)和胰岛素样生长因子- 1 (IGF - 1)的表达。
Objective: To study the effects of exercises training on the improvement of learning and memory ability and the expression of growth associated protein-43 (GAP-43) in vascular dementia rats.
目的：研究运动训练对血管性痴呆大鼠学习记忆功能恢复及组织生长相关蛋白(GAP - 43)表达的影响。
Methods Immunohistochemical technique and hematoxylin eosin stain were used to show the changes in the expression of GAP 43 and pathology.
方法用免疫组化染色法和苏木素伊红染色法研究GAP 43的表达和病理变化的关系。
Conclusion in viral myocarditis, the expression of connexin 43 in the involved cardiac muscle cells was restrained, that may result in dysfunction of gap junction communication.
结论病毒性心肌炎时受累的心肌细胞连接蛋白43的表达受到抑制可能是导致心肌细胞缝隙连接通讯障碍以致心律失常的一个原因。
Objective To study the changes in the expression of growth associated protein 43 (GAP 43) and pathology in photochemically induced temporal cortical infarct of rats and the effects of batroxobin.
目的研究光化学法诱导的颞叶皮层梗死大鼠生长相关蛋白43（GAP 43）的表达及巴曲酶的作用。
CONCLUSION: in myocarditis, expression of connexin 43 and desmin in the involved cardiac muscle cells was inhibited, resulting in dysfunction of gap junctional communication and arrhythmia.
结论：在心肌炎，连接蛋白43和结蛋白表达的参与心肌细胞受到抑制，在间隙连接通讯功能障碍引起的心律失常。
Conclusion in myocarditis, expression of connexin 43 and desmin in the involved cardiac muscle cells was inhibited, resulting in dysfunction of gap junctional communication and arrhythmia.
结论病毒性心肌炎时受累的心肌细胞连接蛋白43和结蛋白的表达受到抑制，可能是导致心肌细胞缝隙连接通讯和收缩功能障碍以致心律失常的一个原因。
Conclusion in myocarditis, expression of connexin 43 and desmin in the involved cardiac muscle cells was inhibited, resulting in dysfunction of gap junctional communication and arrhythmia.
结论病毒性心肌炎时受累的心肌细胞连接蛋白43和结蛋白的表达受到抑制，可能是导致心肌细胞缝隙连接通讯和收缩功能障碍以致心律失常的一个原因。
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