Objective To explore the preventive effect of Weikang granule on gastric ulcer and gastric mucosa damage in rats and its mechanism.
目的探讨胃康颗粒对大鼠胃溃疡及胃黏膜损伤的保护作用及其机制。
The results showed that sodium oleate exerts a significant cytoprotective effect on the gastric mucosa damage induced by indomethacin in a dose related fashion.
油酸钠对消炎痛引起的小鼠胃粘膜损伤有明显的保护作用，并且有剂量—效应关系。
To explore the effect of Buzhong Yiqi Decoction (BYD) on gastrin receptor combination and its protective mechanism for gastric mucosa damage in spleen - deficiency rats.
探讨补中益气汤对脾虚大鼠胃泌素受体结合作用的影响及其对胃粘膜损伤的保护机制。
Objective to study the damage of gastric mucosa in patients with hepatocirrhosis.
目的研究肝硬化并发胃粘膜损伤情况。
Objective To observe the effect of Weichangshu Capsule (WCSC) in inhibiting experimental gastric ulcer and the damage of gastric mucosa in SD rats.
目的研究胃肠舒胶囊抗sd大鼠胃溃疡及抗胃粘膜损伤的作用。
To test the damage degree of gastric mucosa by vernier caliper through the damage model of absolute ethyl alcohol to the SD rats.
采用急性无水乙醇损伤模型，以游标卡尺测定胃黏膜损伤的程度。
Methods The damage of gastric mucosa was observed by using intragastric perfusion with peptone and HCl.
方法采用胃内灌流法观察蛋白胨和不同浓度盐酸对胃粘膜损害作用的影响。
Results: Cu Chl Na Tablets could obviously lessen damage of gastric mucosa and drop incidence rate of gastric ulcer.
结果：能明显减轻胃粘膜损伤、水肿、减少胃溃疡的发生和降低溃疡病的发生率。
The damage degree of gastric mucosa was increased with the elevation of ET level under microscope.
光镜下观察随et水平的升高，胃黏膜损伤程度逐渐加重。
Objective: To study on the effect of Shenxiang Yangwei Capsule (SYC) in inhibiting experimental gastric ulcer and the damage of gastric mucosa in SD rats.
前言：目的：研究参香养胃胶囊抗sd大鼠胃溃疡及抗胃黏膜损伤的作用。
Conclusion ASTmay protect gastric mucosa from ulcer lesion by exhausting exercise, the mechanism may be related to ast raise the gastrin gene expression and against free radical damage.
结论黄芪总苷对运动性疲劳大鼠胃粘膜损伤有保护作用，其保护机制可能与增加胃泌素基因表达，并参与抗氧自由基损伤有关。
Conclusion ASTmay protect gastric mucosa from ulcer lesion by exhausting exercise, the mechanism may be related to ast raise the gastrin gene expression and against free radical damage.
结论黄芪总苷对运动性疲劳大鼠胃粘膜损伤有保护作用，其保护机制可能与增加胃泌素基因表达，并参与抗氧自由基损伤有关。
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