The formation of necrosis cavum and glial scar after spinal cord injured was one of important factor that affected regeneration of nerve fiber and neuron.
脊髓损伤后所形成的坏死空洞和反应性增生的胶质瘢痕是导致脊髓损伤再生修复受阻的重要因素。
After brain damage, the expression of neurocan obviously increases again in the glial scar. It is considered that neurocan can prevent the regeneration of axon.
脑损伤后，神经蛋白聚糖在胶质瘢痕中的表达明显增加，据认为可阻止轴突再生。
After sacrifice, spinal cord sections were analyzed for MSC graft size, tissue sparing, host immune response, and glial scar formation, using specific antibodies and Nissl-myelin staining.
处死后，用特殊的抗体和髓鞘尼氏染色法处理脊髓切片，分析骨髓基质细胞移植物大小、组织保留情况、宿主免疫反应和神经胶质疤痕形成情况。
After sacrifice, spinal cord sections were histologically analyzed for presence of graft-derived immune cells, host immune response, tissue sparing, glial scar formation, and grafting efficacy.
处死后，进行脊髓切片病理分析移植性免疫细胞存在情况，宿主的免疫反应情况，组织保留情况，胶质瘢痕形成情况和移植治疗效果。
After sacrifice, spinal cord sections were histologically analyzed for presence of graft-derived immune cells, host immune response, tissue sparing, glial scar formation, and grafting efficacy.
处死后，进行脊髓切片病理分析移植性免疫细胞存在情况，宿主的免疫反应情况，组织保留情况，胶质瘢痕形成情况和移植治疗效果。
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