To observe the effects of Vitamin K3 (VK3) on cell growth and apoptosis in human ovarian cancer cell line SKOV3 and to explore its mechanism.
观察维生素k3 (VK3)对人卵巢癌细胞株skov3增殖和凋亡的影响，并探讨其作用机制。
This article evaluates the effect of heparanase gene on the proliferation, adhesion and invasion of human ovarian cancer cell line SKOV3 cells.
本文探讨乙酰肝素酶基因转染卵巢癌SKOV3细胞后对细胞生长、黏附、侵袭能力的影响。
Objective: to investigate the influence of TOFA, a Acetyl CoA Carboxylase inhibitor, on cell growth of human ovarian cancer cell line COC1, and explore the mechanism.
目的：探讨乙酰辅酶A羧化酶抑制剂TOFA对卵巢癌COC1细胞增殖能力的抑制作用及其机制。
Objective:To observe caesalpinia sappan aqueous extract's growth inhibiting effects and investigated the mechanism of apoptosis-inducing on human ovarian cancer cell line SKOV3.
目的：观察苏木水提物对人卵巢癌细胞SKOV3的生长抑制作用，探讨其诱导靶细胞凋亡及作用机制。
This study was to observe the effects of 2-chloro-adenosine(2-ClAdo) , 2-chloro-2-deoxyadenosine (2-CldAdo), and 2-deoxyadenosine (2-dAdo ) on invasion of human ovarian cancer cell line HO-8910PM.
本研究拟观察2-氯腺苷、2-氯脱氧腺苷、2'-脱氧腺苷对人高转移卵巢癌细胞HO-8910PM体外侵袭的抑制作用。
VEGF-C in human ovarian promoted tumor cell proliferation, invasion and metastasis, was related to epithelial ovarian cancer prognosis.
VEGF- C诱导卵巢肿瘤细胞的增殖，促进侵袭及转移，与上皮性卵巢肿瘤的预后有关。
Using nude mice with established xenografts of human ovarian cancer, LDN was found to repress tumor progression, reducing DNA synthesis and angiogenesis but not altering cell survival.
结果发现LDN可以抑制肿瘤的增生，降低去氧核糖核酸合成以及血管新生，但不改变细胞的存活率。
They found these stem cells, which act as a kind of master cell, first in batches of ovarian tumor cells taken from mice. They then identified similar cells in human cancer cells.
他们发现这些细胞扮演着一种基本细胞的角色，首先从小鼠的卵巢肿瘤分离得到，而后又在人的癌细胞中鉴定出类似的细胞。
Conclusion: MTS1 is able to disturb the cell cycle of HO-8910 and suppress the cell division. This provides a further evidence for gene therapy of MTS1 on human ovarian cancer.
结论：MTS1基因可以改变卵巢癌细胞HO- 8910的细胞周期，这可能为卵巢癌的基因治疗提供了新的实验依据。
Conclusion: MTS1 is able to disturb the cell cycle of HO-8910 and suppress the cell division. This provides a further evidence for gene therapy of MTS1 on human ovarian cancer.
结论：MTS1基因可以改变卵巢癌细胞HO- 8910的细胞周期，这可能为卵巢癌的基因治疗提供了新的实验依据。
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