The impacts of L-NAME on hypoxic pulmonary hypertension were studied.
研究l -NAME对缺氧性肺动脉高压的影响。
AIM: To study the role of adrenomedullin (AM) in the pathogenesis of hypoxic pulmonary hypertension.
目的：探讨肾上腺髓质素(AM)在慢性低氧性肺动脉高压发病中的作用。
Objective: To study the pattern of right ventricle remodeling in hypoxic pulmonary hypertension rats.
目的：研究缺氧性肺动脉高压大鼠右心室重构情况。
Purpose To study the effects of Endothelin-1 (ET-1) in hypoxic pulmonary hypertension (HPH) and its relationship with HPH.
目的：探讨内皮素- 1 (et - 1)与缺氧性肺动脉高压(HPH)的相互关系。
Objective To study the effect of local pulmonary arterial renin angiotensin system on hypoxic pulmonary hypertension (PAH).
目的探讨肺动脉局部肾素-血管紧张素系统对缺氧性肺动脉高压(PAH)的影响。
Conclusion : Eccentric hypertrophy accounts for the early change of right ventricle in hypoxic pulmonary hypertension rats.
结论：缺氧性肺动脉高压大鼠右心室早期表现为离心性肥大。
However, in hypoxic pulmonary hypertension rats, 5 - HT - induced vasoconstriction is mediated by both the 5 - HT1Band 5 - HT2A receptor.
但是，慢性低氧肺动脉高压大鼠5-HT诱导的血管收缩反应是通过5 -HT _ (2 A)和5 - HT _ (1 B)受体介导的。
Conclusion Nitric oxide release decreased during chronic hypoxia, which was probably involved in the mechanism of hypoxic pulmonary hypertension.
结论大鼠慢性缺氧时一氧化氮释放减少，而一氧化氮释放减少可能参与缺氧性肺动脉高压的形成。
These results proved that polycythemia may play an important role in pathogenesis of chronic hypoxic pulmonary hypertension and cardiac hypertrophy.
以上结果表明红细胞增多可能在慢性缺氧性肺动脉高压和右室肥大发生起着重要作用。
The above results proved that polycythemia might play an important role in pathogenesis of hypoxic pulmonary hypertension and myocardial hypertrophy.
以上结果表明，红细胞增多在缺氧性肺动脉高压和右室肥大中起着重要作用。
Objective the aim of this study was to examine the modulatory role of endogenous nitric oxide in the development of chronically hypoxic pulmonary hypertension.
目的探讨一氧化氮对慢性缺氧性肺动脉高压的调节作用。
Hypoxic pulmonary hypertension (HPH) is a key link of the adaptation physiology in high altitude area and the pathogenesis of all types of high altitude diseases.
低氧性肺动脉高压(HPH)是高原适应生理的重要环节，也是各型高原病的发病机理。
Objective To evaluate the value of acoustic densitometry (AD) in detecting myocardium damage of the left ventricle secondry to hypoxic pulmonary hypertension (PH).
目的探讨声学密度定量（AD）技术定量评价缺氧肺动脉高压（PH）后左室心肌损害的应用价值。
Conclusion: the endoplasmic reticulum stress-induced apoptosis may be one of the mechanism of hypoxic pulmonary hypertension and pulmonary vascular wall remodeling.
结论：内质网应激介导的凋亡与大鼠慢性低氧性肺动脉高压、肺血管改建的病理过程有关。
Conclusion: Hypoxia induces elevated secretion of VEGF and the changes of VEGF may be related to hypoxic pulmonary hypertension, hypoxic adaptative regulation and complications of HAPC.
结论：高原缺氧诱导了VEGF分泌增多，VEGF在HAPC的肺动脉高压、低氧适应调节及并发症中可能发挥了重要作用。
Conclusions: the CTGF may play an important role in the pathogenesis process of hypoxic pulmonary vascular remodeling and pulmonary hypertension.
结论低氧所致CTGF的合成增多在低氧性肺血管重建和肺动脉高压的发病过程中起一定的作用。
AIM: To explore the effect of exogenous carbon monoxide (CO) on the hypoxic pulmonary arterial hypertension in the rat.
目的：探讨外源性低浓度一氧化碳(CO)在低氧性肺动脉高压中的作用。
Objective To investigate the effect of carbon monoxide (CO) inhalation on acute hypoxic pulmonary arterial hypertension and acute hypoxic lung injury.
目的研究吸入一氧化碳(CO)对急性缺氧性肺动脉高压及缺氧性肺损伤的影响。
AIM: To investigate changes of carbon monoxide (CO) in plasma and tissue during acute hypoxia and effect of exogenous CO on acute hypoxic pulmonary arterial hypertension.
目的：观察急性缺氧时血浆、组织内源性一氧化碳的变化及外源性一氧化碳对急性肺动脉高压的影响。
AIM: to evaluate the prophylactic effect of aminophylline on the pulmonary hypertension rebound reaction to exposure to no in the hypoxic pigs.
目的：评价氨茶碱对缺氧性动物吸入NO肺动脉高压反弹的预防作用。
AIM: to evaluate the prophylactic effect of aminophylline on the pulmonary hypertension rebound reaction to exposure to no in the hypoxic pigs.
目的：评价氨茶碱对缺氧性动物吸入NO肺动脉高压反弹的预防作用。
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