AIM In order to study the blocking effects of extracellular Li + on the inward rectifier potassium channel (IRK1 T) and its mechanism.
目的研究 L i+对非洲爪蟾卵母细胞表达的双基因内向整流钾通道 （IRK1- T）的作用及其机制 。
Results Inward rectifier characteristics of inward rectifier potassium channel current were weak when Mg 2+ was absent, whereas strong when Mg 2+ was present.
目的探讨镁离子对豚鼠心室肌细胞内向整流钾通道内向整流作用的影响。
CONCLUSION: Hypercholesterolemia altered the vascular functions and regulated gene expression levels of specific inward rectifier and ATP-sensitive potassium channel subtypes.
结论：高胆固醇血症可改变血管功能，并影响某些内向整流和AT P敏感钾通道亚型的基因表达。
CONCLUSION: Hypercholesterolemia altered the vascular functions and regulated gene expression levels of specific inward rectifier and ATP-sensitive potassium channel subtypes.
结论：高胆固醇血症可改变血管功能，并影响某些内向整流和AT P敏感钾通道亚型的基因表达。
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