Conclusions ET-1 is perhaps participated in limb ischemia-reperfusion Injury.
结论ET - 1可能参与了肢体缺血再灌注损伤。
Conclusion: CVB-D has protective effect on cerebral ischemia-reperfusion injury in rats.
结论：CVB - D对大鼠局灶性脑缺血再灌注损伤具有保护作用。
It is not reported that the protective effects of Imi on cerebral ischemia-reperfusion injury.
目前，该药对整体脑缺血-再灌注损伤保护作用的研究尚未见报道。
Oxygen free radical may play important roles in myocardial ischemia-reperfusion injury after CPB.
氧自由基在CPB后心肌缺血再灌注损伤中起重要作用。
Objective: To observe the effect of Eleulheroside in protecting gastric ischemia-reperfusion injury.
目的：观察刺五加对胃缺血再灌注损伤的防护作用。
Conclusions Complement and inflammatory reaction participated in cerebral ischemia-reperfusion injury.
结论补体及炎症反应参与脑组织缺血再灌注损伤。
Methods: a model of acute ischemia-reperfusion injury was produced in open chest anaesthetized rabbits.
方法：采用麻醉开胸兔急性心肌缺血再灌注损伤模型。
Objective: To observe the protective effect of tea saponin on Focal cerebral ischemia-reperfusion injury.
目的观察茶皂素对大鼠局灶性脑缺血再灌注损伤的保护作用。
Objective To study the protection of ischemic preconditioning on the hepatic ischemia-reperfusion injury.
任智目的评价缺血预处理对大鼠肝脏的保护作用。
So it is concluded that hemodilution has distinct curative effect on myocardial ischemia-reperfusion injury.
提示血液稀释对心肌缺血及再灌注损伤有明显的治疗作用。
Methods Rat model of myocardial ischemia-reperfusion injury was prepared by ligating the left coronary artery.
采用左冠状动脉结扎的方法复制大鼠缺血再灌注损伤模型。
Objective to observe the role of mitochondria dysfunction in neuron apoptosis during ischemia-reperfusion injury.
目的观察线粒体功能失调在缺血再灌后神经元凋亡中的作用。
Objective To study the protective effect of hyperoxic liquid preconditioning in myocardial ischemia-reperfusion injury.
目的探讨高氧液预处理对兔心肌缺血再灌注损伤的保护作用。
To investigate the protective effect and mechanism of dipyridamole against ischemia-reperfusion injury on rabbit liver.
探讨潘生丁对兔肝脏热缺血再灌注损伤的保护作用及其相关机制。
Objective: To study the effects of L-carnitine on myocardial ischemia-reperfusion injury and energy metabolism in rats.
目的：观察左旋卡尼汀对离体大鼠心脏缺血-再灌注损伤的作用及对心肌细胞能量代谢的影响。
Objective To explore the protective effect of baicalin on the cerebral ischemia-reperfusion injury in rats and its mechanism.
目的观察黄芩苷对大鼠脑缺血再灌注损伤的保护作用，并初步探讨其作用机制。
Objective To evaluate the effect of ligustrazine and ulinastatin on protecting ischemia-reperfusion injury after limb replantation.
目的：评价川芎嗪和乌司他丁对断肢再植缺血再灌注损伤的保护作用。
Objective To explore the feasibility of neural stem cells (NSCs) transplantation for focal brain ischemia-reperfusion injury in rats.
目的探讨大鼠胚胎神经干细胞移植治疗局灶性脑缺血再灌注损伤的可行性。
Objective To study the effects and mechanism of Naotaifang extract preconditioning on the cerebral ischemia-reperfusion injury in gerbil.
目的：研究脑泰方提取物预处理减轻沙土鼠脑缺血再灌注损伤的作用及作用机理。
Objective To study the protective effect of breviscapine injection on microcirculation of ischemia-reperfusion injury in mesentery of rabbit.
目的探讨灯盏花素注射液对家兔缺血再灌注损伤肠系膜微循环的保护作用。
OBJECTIVE To investigate the protective effects of M3 receptor agonist pilocarpine on myocardial ischemia-reperfusion injury in isolated rat hearts.
目的研究M3受体激动剂毛果芸香碱对离体大鼠心肌缺血再灌注损伤的保护作用。
Ischemic preconditioning 24 hours before ischemia-reperfusion injury increased VEGF-A levels and substantially decreased the number of apoptotic retinal cells.
在缺血-再灌注损伤前24小时进行缺血的预处理可增加VEGF - A水平并大量减少凋亡的视网膜细胞数量。
These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.
这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Conclusion rat retina ischemia-reperfusion injury can be observed after ligating common carotid artery. Apoptosis may play an important role in rat retina ischemia-reperfusion injury.
结论通过结扎颈总动脉，可以见到大鼠视网膜的缺血再灌注损伤，而细胞凋亡在视网膜缺血再灌注损伤中发挥着重要作用。
It is important to reduce ischemia reperfusion injury.
重要的是要降低缺血再灌注损伤。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Objective To clarify the effect of nitrous oxide for the treatment of spinal cord ischemia reperfusion injury.
目的研究一氧化氮在脊髓缺血再灌注损伤中的作用。
Objective To clarify the effect of nitrous oxide for the treatment of spinal cord ischemia reperfusion injury.
目的研究一氧化氮在脊髓缺血再灌注损伤中的作用。
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