Objective: to study protective mechanisms of superficial needling to ischemic penumbra neuron in cerebral infarction rats.
目的：研究浅刺针法对脑梗塞大鼠脑组织缺血半暗带神经细胞的保护作用及其机制。
New imaging techniques are helpful in identifying ischemic penumbra and the selection of patients for thrombolytic therapy.
新的影像学技术有助于判断缺血半暗带和选择适合溶栓的病例。
The identification of these patients is of importance because the patients with ischemic penumbra are more like ly to benefit from the treatment.
由于存在缺血半暗带的患者更有可能从治疗中获益，因此识别这些患者至关重要。
ResultS COX - 2 protein induction paralleled temporally and overlapped anatomically the appearance of TUNEL - positive cells in ischemic penumbra.
结果环氧酶-2蛋白的表达与TUNEL阳性细胞在时程上一致，解剖部位上重叠。
On the early onset, thrombolytic therapy could timely restore blood flow of ischemic tissue and efficiently salvage the function of ischemic penumbra.
血栓溶解疗法可以迅速恢复缺血脑组织的血流，能有效挽救缺血半暗带的功能。
The therapeutic target is to salvage potentially reversible ischemic tissue (ischemic penumbra, IP) to reduce infarct size and improve neuromuscular function.
其治疗目标应是挽救可逆性缺血组织—缺血性半暗带(IP)，以缩小梗塞范围和改善神经功能。
To recover blood supply of brain tissue in ischemic region especially in ischemic penumbra in time, it will have great significance in treating and prognosis.
脑缺血后，及时恢复缺血区脑组织的血液供应，尤其是缺血半暗带区的血液供应，对于缺血性脑血管病的治疗与预后意义重大。
The article reviews the changes of hematoma and ischemic penumbra, brain edema, hyperfibrinolysis after cerebral hemorrhage, and as well as amyloid angiopathy.
文章就脑出血后的血肿变化和缺血半暗带、脑水肿和纤溶亢进以及血管淀粉样变等进行了综述。
Aim: To investigate the changes of amyloid precursor protein (APP) transcriptional level at different ischemic times in rats' ischemic penumbra and core of cortices.
目的：研究大鼠局灶性脑缺血不同缺血时间皮质半暗带和中心区淀粉样前体蛋白(APP)在转录水平表达规律。
Objective To explore the value of diffusion weighed imaging (DWI) and perfusion weighted imaging (PWI) in the diagnosis of the ischemic penumbra (IP) of superacute infarction.
目的探讨磁共振扩散加权成像（DWI）和灌注加权成像（PWI）对早期脑梗死缺血半暗带（IP）的诊断价值。
Where ischemic penumbra existed, the intra-arterial thrombolysis was administered. It was assessed by NIHSS score before and after thrombolytic therapy at the 3rd week and the 3rd month.
对存在缺血半暗带患者给予动脉溶栓治疗，并分别于溶栓前、溶栓后即刻、溶栓后3周和3个月进行NIHSS评分，以评估近期及远期疗效。
Then to determinate NG2 of O4 positive cells in the ischemic core, ischemic penumbra and contralateral area of rats at each time-point after reperfusion by immunofluorescence histochemistry.
用免疫荧光组织化学法检测脑缺血再灌注后早期大鼠大脑梗死中心区、梗塞周边区和缺血对侧NG2和O4阳性细胞数量。
Results Well defined ischemic core and penumbra area appeared in each experimental animal.
结果小鼠均出现成熟缺血核心区和半暗区。
The clinical treatment of the key is to save the ischemic region (penumbra) of dying neurons and promote damage nerve function recovery.
临床上治疗的关键在于挽救缺血区域（半暗带）的濒临死亡的神经元和促进损伤后神经功能的恢复。
CONCLUSION: GLUT1 expression is notably up-regulated in the penumbra region after focal cerebral ischemia, it may be a protective reaction against ischemic injury.
结论：GLUT1在缺血半影区的表达上调，可能是机体对缺血损伤的保护性反应。
Whereas cPKC II and membrane translocation levels in penumbra of ischemic cortex in H4+Ischemia group increased significantly compared with that of mice in H0+Ischemia group (p<0.05).
与H0缺血组小鼠皮层半影区相比，H4缺血组小鼠皮层半影区内PKCII和膜转位水平明显增高 （p<0.05）。
Whereas cPKC II and membrane translocation levels in penumbra of ischemic cortex in H4+Ischemia group increased significantly compared with that of mice in H0+Ischemia group (p<0.05).
与H0缺血组小鼠皮层半影区相比，H4缺血组小鼠皮层半影区内PKCII和膜转位水平明显增高 （p<0.05）。
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