All these cases had acute hemolytic anemia, jaundice, hemoglobinuria and reduced G6PD activity to different extents.
所有患者都具有急性溶血性贫血，黄疸等临床表现及不同程度的血红蛋白尿、G6PD酶活性降低。
The clinical manifestations of these subjects were acute hemolytic anemia trigged by ingestion of fava bean and neonatal jaundice.
临床主要表现为新生儿黄疸、进食蚕豆后发生急性溶血性贫血等。
Our Studies suggest that all the gene mutation may le AD to jaundice of the newborn, acute hemolytic anaemia, and have some relation to viral hepatitis, leukaemia, lymphoma.
研究发现，各型基因突变均可引起新生儿高胆红素血症、急性溶血性贫血等，也与病毒性肝炎、白血病、淋巴瘤等疾病的发生有一定关系。
Objective: To study the relations between the blood type antibody titer of 232 blood-type-O gravidas and the occurrences of hemolytic disease and jaundice among newborn infants.
目的：探讨232例O型血孕妇血型抗体效价与新生儿溶血病及高胆红素血症发生的关系。
Objective: To study the relations between the blood type antibody titer of 232 blood-type-O gravidas and the occurrences of hemolytic disease and jaundice among newborn infants.
目的：探讨232例O型血孕妇血型抗体效价与新生儿溶血病及高胆红素血症发生的关系。
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