Methods: Bovine lens epithelial cells (BLEC) were cultured in vitro.
方法：牛晶体上皮细胞原代培养。
Conclusion High power EMP radiation inhibited proliferation of lens epithelial cells.
结论高场强大剂量的EMP辐射可抑制晶状体上皮细胞的正常增殖。
RT-PCR was applied to identity the expression of FGFR1 mRNA in rat lens epithelial cells.
采用剥取晶状体囊膜组织方法，应用RT-PCR技术检测FGFR1在转录水平的表达。
Objective: To investigate the DNA damage in the lens epithelial cells in cataract patients.
目的：欲研究白内障病人晶状体上皮细胞是否存在DNA损伤及损伤的程度。
Results 0.25%? 0.2% trypsin can clean up lens epithelial cells from anterior capsules totally.
结果0 .2 5 %、0 .2 %胰蛋白酶溶液处理的晶状体前囊，晶状体上皮细胞可以完全脱落。
Results the positive expression of CLC-3 was distributed in membrane of the lens epithelial cells.
结果CLC - 3阳性表达于晶状体上皮细胞的细胞膜。
ConclusionAdding lens cortex can increase the success rate of primary culture of lens epithelial cells.
结论添加晶体皮质可提高晶体上皮细胞原代培养成功率。
To investigate the biological characteristics of human embryonic lens epithelial cells cultured in vitro.
目的观察体外培养人胚晶状体上皮细胞的生物学特性。
There is no direct relationship between the expression of LEP503 and the proliferation of lens epithelial cells;
LEP503基因与晶状体上皮细胞增生没有直接关系；
Objective: to observe the expression of epidermal growth factor (EGF) in lens epithelial cells (LECs) of human fetus.
前言：目的：观察表皮生长因子(EGF)在人胎儿晶状体上皮细胞的表达。
AIM: to investigate the effect of high glucose with various concentration on apoptosis of human lens epithelial cells.
目的：研究不同浓度的高糖对人晶状体上皮细胞凋亡的作用。
AIM: To study the effect of heat shock protein-70 (HSP-70) on lens epithelial cells (LECs) in human diabetic cataract.
目的：为探讨热休克蛋白- 70对人类糖尿病性白内障晶状体上皮细胞的影响。
Objective To investigate the expression of heat shock protein 27 (HSP27) in human age related cataract lens epithelial cells.
目的探讨热休克蛋白(HSP)27在老年性白内障晶状体上皮细胞中的表达及意义。
Expression of Matrix Metalloproteinase-2, -9 and Their Inhibitors TIMP-1, -2 on Lens Epithelial Cells of Aged-related Cataract.
基质金属蛋白酶-2,9及其抑制因子- 1,2在老年性白内障晶状体上皮细胞的表达。
Methods: Using the developed procedure to culture rabbit lens epithelial cells, and the cultured cells were examined morphologically.
方法：利用改良消化法对兔晶体上皮细胞进原代培养，对培养的细胞进行形态学观察。
Objective to study the inhibitory effect of disintegrin on adhesion and migration in cultured human lens epithelial cells (LECs) in vitro.
目的研究去整合素对体外培养的人眼晶状体上皮细胞生长的抑制作用。
Theoretically, clearance of the remaining lens epithelial cells within the capsular bag with medicine is the most effective way to prevent PCO.
理论上说，使用药物彻底清除残留于晶状体囊袋内的晶状体上皮细胞将是预防后发性白内障最直接的方法。
Methods New Zealand white rabbit lens epithelial cells were primarily Cultured. After the confluence, the cells were trysinized and sub-cultured.
方法：取新西兰白色家兔晶体上皮细胞进行原代培养，细胞融合后用胰蛋白酶进行消化传代。
The opacification is mainly due to the proliferation and migration of postoperative remnants of lens epithelial cells on the posterior lens capsule.
残留晶状体上皮细胞在后囊的增殖、移行、纤维化生是后囊混浊形成的重要因素。
PCO is caused by the lens epithelial cells retained in the capsular bag following surgery, which proliferate, migrate and transform to myofibroblasts.
术后晶状体囊残留的晶状体上皮细胞的增殖、迁移、纤维化生是形成后发性白内障的主要原因。
But till up now it was rarely known for the mechanism of contusion cataract, so was the accurate report for the ultrastructure of lens epithelial cells.
但是迄今硕士研究生学位论文为止，对钝挫伤白内障发病机制的研究尚不多见，也未见到其晶状体上皮细胞凋亡超微结构的明确报道。
ResultsBy DNA fragmentation assay, the DNA "ladder" were found in senile cataract lens epithelial cells, while no DNA "ladder" were found in normal cells;
结果DNA片段分析显示，老年性白内障患者晶状体上皮细胞有DNA“梯状”图谱，而健康成年人晶状体上皮细胞未见DNA 断裂现象；
For many years, many scholars of domestic and overseas had been committed to study the methods to suppress the proliferation of lens epithelial cells by drugs.
多年来国内外学者一直致力于通过药物等方法抑制晶状体上皮细胞增殖的研究。
Conclusions HPM may cause lens injury and cataract formation. HPM-induced proliferation disorder in lens epithelial cells may possesses a critical role in cataract development.
结论H PM可导致晶状体损伤及白内障发生，辐射引起上皮细胞增殖异常是白内障发生的重要原因。
Objective To study the pathogensis of lens posterior capsular opacification and the effects of different ocular tissue on bovine lens epithelial cells(BLEC) in cell culture level.
目的从细胞水平探讨后囊混浊形成的机理，及各种眼组织对晶状体上皮细胞的作用。
Objective To investigate the changes and the role of telomerase activity and other stress-related proteins in the process of UV-induced DNA damage and repair in human lens epithelial cells.
目的探讨在紫外线诱导的人晶状体上皮细胞DNA损伤修复过程中端粒酶活性和氧化损伤蛋白的变化和作用。
The results revealed that the in vitro survival capacity of the human lens epithelial cells is limited and the proliferative capacity of the cells is negatively cor-related to the donor age.
结果显示，人类晶体上皮细胞在体外生存能力有限，体外的生长能力与供体年龄呈负相关。
Immunohistochemical techniques and computer-imaging analysis were used to localize and observe the expression of AQP1 in lens subcapsule epithelial cells.
应用免疫组织化学方法和计算机图像分析技术对前囊膜下晶状体上皮细胞AQP1进行检测。
Immunohistochemical techniques and computer-imaging analysis were used to localize and observe the expression of AQP1 in lens subcapsule epithelial cells.
应用免疫组织化学方法和计算机图像分析技术对前囊膜下晶状体上皮细胞AQP1进行检测。
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