PAMP include lipopolysaccharide (LPS), lipoteichoicacid, peptidoglycan, and teichoic acid mannose.
PAMP包括脂多糖（LPS）、脂磷壁酸、肽聚糖和甘露糖等。
Objective To observe the preventive and roductive effect of Chaishao Chengqi Decoction (CSCQ)in intussusception mice model induced by lipopolysaccharide(LPS).
目的利用脂多糖小鼠肠套叠动物模型观察柴芍承气汤对肠套叠的预防及还纳作用。
OBJECTIVE To study the effect of fresh herba houttuyniae extract on lipopolysaccharide(LPS)-induced lung inflammation in mice.
目的研究鲜鱼腥草提取物对脂多糖所致小鼠肺部炎症的影响。
OBJECTIVE to investigate the regulative role of WuLing capsules on liver injuries caused by hepatic cytokine induced by lipopolysaccharide (LPS).
目的观察五灵胶囊对脂多糖(LPS)诱导肝脏内细胞因子所致肝损伤的调节作用。
Objective: the apoptosis of human umbilical vascular endothelial cell (HUVEC) was induced by lipopolysaccharide (LPS) in order to explore syndrome of blood stasis and its molecular mechanism.
目的：从脂多糖(LPS)所致血管内皮细胞(vec)凋亡的角度进行了实验研究，为进一步探讨血瘀证实质及分子机制。
Objective: to investigate the effect of cholecystokinin octapeptide (CCK8) on lipopolysaccharide (LPS) induced apoptosis of cultured pulmonary artery endothelial cells (BPAEC).
目的：探讨八肽胆囊收缩素(CCK 8)对脂多糖(LPS)诱导培养的肺动脉内皮细胞(BPAEC)凋亡的影响。
Objective To explore the effect and mechanism of lipopolysaccharide (LPS) on rat gastric mucosal cells line (RGM-1).
目的探讨内毒素脂多糖(LPS)对胃粘膜上皮细胞rgm - 1的作用及其机制。
Endotoxin lipopolysaccharide (LPS) induces thrombosis by stimulating PAI-1 secretion of vascular cells (ec).
内毒素脂多糖(LPS)通过刺激血管内皮细胞(EC)分泌PAI - 1且而导致血栓形成。
ABSTRACT: Objective to investigate the effects of emodin on expression of cytokines induced by lipopolysaccharide (LPS) in cultured human corneal fibroblasts in vitro.
摘要：目的观察大黄素对脂多糖(LPS)诱导的角膜基质细胞表达细胞因子的影响。
Objective: To establish a isolation and cultivation method of mice lung microvessel endothelial cell (LMVEC) and investigate the VEGF expression of mice LMVEC induced by lipopolysaccharide (LPS).
目的：研究小鼠肺微血管内皮细胞分离培养方法并探讨脂多糖(LPS)诱导小鼠肺微血管内皮细胞血管内皮生长因子(VEGF)表达。
Objective To investigate the anti-inflammatory effects of secretory leukocyte protease inhibitor(SLPI) on newborn rats with lipopolysaccharide(LPS)-induced acute lung injury(ALI).
目的探讨分泌型白细胞蛋白酶抑制剂（SLPI）对内毒素（LPS）致新生大鼠急性肺损伤（ALI）的抗感染作用。
Objective: To investigate the expression of CXCL 12 and its potential functions in neonatal rat lung caused by lipopolysaccharide (LPS).
前言：目的：研究内毒素作用于新生大鼠后，肺组织趋化因子-基质细胞衍生因子(CX CL12)表达的变化及可能作用。
Conclusion There was no response of HICs to LPS stimulation, and no expression of TLR4, CD14 and MD-2 on HICs may be an important molecular mechanism by which they tolerated to lipopolysaccharide.
结论HIC呈内毒素无反应性，其表面内毒素相关受体tlr4、CD 14和MD 2不表达是其耐受内毒素作用的重要分子机制。
Objective To study the directly injury effects of lipopolysaccharide(LPS) on human vascular endothelial cells(HVEC).
目的探讨脂多糖（LPS）对人血管内皮细胞（HVEC）直接损伤作用。
Objective To evaluate the effect of drug on chemical degradation of lipopolysaccharide (LPS) from oral anaerobes.
目的评估各种药物对口腔厌氧菌内毒素的降解作用。
Aim To investigate the effect of Dicranostiga Leptodu (Maxim) Fedde (DLF) on experimental liver injury induced by Calmette Guerin bacillus vaccine (BCG) and Lipopolysaccharide (LPS) in mice.
目的探讨秃疮花提取物（DLF）对卡介苗（BCG）和脂多糖（LPS）诱导的小鼠免疫性肝损伤的影响。
They first showed that stimulation with lipopolysaccharide (LPS) and concanavalin a increased tonsillar and adenoidal proliferation in cells from OSA children compared with controls.
他们首先显示出在脂多糖和刀豆蛋白a的刺激下，OS A儿童较糖皮质激素控制组的扁桃体和腺样体的细胞增殖增多。
This review describes the signaling transduction of TLR4, the relationships between TLR4 and lipopolysaccharide(LPS), TLR4 and diseases, confirms the significance of TLR4 research.
本文就TLR4的信号转导、TLR4与LPS的关系进行了综述，并介绍了TLR4与相关疾病之间的关系，对TLR4的研究意义进行了肯定。
AIM: to study the effect of cholecystokinin octapeptide (CCK-8) on lipopolysaccharide (LPS) -stimulated pulmonary interstitial macrophage (im) in vitro.
目的：初步探讨八肽胆囊收缩素(CCK - 8)对体外脂多糖(LPS)激活大鼠肺间质巨噬细胞(IM)的调节作用。
We hypothesize that reactive oxygen species (ROS) mediate lipopolysaccharide (LPS) -evoked hepatic lipid accumulation.
我们假定活性氧簇分子参与了脂多糖诱导的肝细胞脂肪堆积。
Objective To study the protective effect of propofol on acute lung injury (ALI) induced by lipopolysaccharide (LPS) in rats.
目的研究丙泊酚对内毒素(LPS)诱导的大鼠急性肺损伤(ALI)的保护作用及其可能分子作用机制。
Objective to investigate the influence of lipopolysaccharide (LPS) on macrophages expressing TNF-alpha related apoptosis induced-ligand (TRAIL) and its relation to apoptosis of HepG2 cell line.
目的探讨脂多糖(LPS)、巨噬细胞表达肿瘤坏死因子相关凋亡诱导配体(TRAIL)和肝细胞凋亡的相互关系。
Objective:To investigate the effect of Lipopolysaccharide(LPS)on intra-uterine fetal death(IUFD), fetal growth retardation(IUGR)and skeletal development retardation in mice.
目的：研究细菌脂多糖（LPS）对小鼠宫内胎儿死亡（IUFD）、生长发育迟缓（IUGR）和骨骼发育迟缓的影响。
Objective:To investigate the effect of Lipopolysaccharide(LPS)on intra-uterine fetal death(IUFD), fetal growth retardation(IUGR)and skeletal development retardation in mice.
目的：研究细菌脂多糖（LPS）对小鼠宫内胎儿死亡（IUFD）、生长发育迟缓（IUGR）和骨骼发育迟缓的影响。
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