To prove the lung adenocarcinoma cells oncogenic capability in vitro.
并初步证实肺腺癌细胞体外致瘤能力的差异性。
To investigate the suppression effect of tumor suppressor gene in lung adenocarcinoma cells.
探讨肿瘤抑制基因对肺腺癌细胞生长的抑制作用。
Objective to study the DNA damage and repair of normal lung interstitial cells and human lung adenocarcinoma cells exposed to cigarette smoke.
目的探讨经香烟烟雾溶液染毒的人正常肺间质细胞和人肺腺癌细胞的DNA损伤及其修复效应。
Conclusion: Transfection of DTA gene in vitro may kill human lung adenocarcinoma cells and reduce the oncogenicity of human lung adenocarcinoma cells.
结论体外转染白喉毒素a基因可杀伤人肺腺癌细胞并降低人肺腺癌细胞的致瘤性。
Objective to investigate the effects of ionizing radiation (IR) on vascular endothelial growth factor (VEGF) expression in human lung adenocarcinoma cells.
目的探讨电离辐射对人肺腺癌细胞体外表达血管内皮生长因子(VEGF)的影响。
Conclusion Aspirin can inhibit the proliferation of lung adenocarcinoma cells A549 through influencing the distribution of cell cycle and inducing apoptosis of A549 cells.
结论阿司匹林可抑制肺腺癌细胞A549的增殖，其影响细胞周期分布、诱导细胞凋亡可能是其重要的机制。
Conclusion The overexpression of CC10 might play an important role in the matrine-induced apoptosis of lung adenocarcinoma A549 cells.
结论CC10基因表达增高可能在苦参碱诱导肺腺癌A549细胞凋亡中起重要作用。
AIM: to study the apoptosis of human lung adenocarcinoma A549 cells induced by CN-2, a derivative of podophyllotoxin, and to obtain insights into its molecular mechanism of action.
目的研究鬼臼素衍生物cn - 2对人肺腺癌细胞A549的凋亡诱导作用，并对其分子机制进行初步探讨。
Objective To investigate the chemosensitivity of irradiated resistant cells of multicellular spheroids in A549 lung adenocarcinoma.
目的探讨A549肺腺癌多细胞球体放射抗拒细胞的化疗药物敏感性。
Moreover, the unity of MMP-2 and VEGF immunocytochemistry is helpful for pathological diagnosis of adenocarcinoma cells of lung cancer.
两者联合做免疫细胞化学检查对肺腺癌细胞病理诊断有辅助意义。
We know mostly the adenocarcinoma cells are from the lung. However, the chance of second primary is still present.
我们知道多数腺癌细胞来自肺脏，但仍然有第二原发灶存在的可能。
Objective To investigate the roles of PI3K pathway in multicellular resistance of lung adenocarcinoma A549 cells.
目的探讨PI3K信号通路在肿瘤群集耐药中的作用。
Objective To investigate the roles of PI3K pathway in multicellular resistance of lung adenocarcinoma A549 cells.
目的探讨PI3K信号通路在肿瘤群集耐药中的作用。
应用推荐