The rats after aorta coarctation had severe hypertension and myocardial hypertrophy.
研究发现腹主动脉狭窄后大鼠发生了严重的高血压及心肌肥厚。
The left ventricular myocardial hypertrophy was not significant in the mild hypertension patients.
而在轻度组患者，左室心肌肥厚不显著；
Objective: To study the role of nitric oxide in myocardial hypertrophy caused by pressure overload in rats.
目的：探讨一气化氮在压力超负荷心肌肥大反应过程中的作用。
Objective To discuss the effects of rennin-angiotensin system on myocardial hypertrophy in simple obesity children.
目的探讨单纯性肥胖儿童肾素血管紧张素系统与心肌肥厚的关系。
In he stain, myocardial hypertrophy, severe myolysis and extensive fibrosis in the interstitial space of the atria were found in AF group.
HE染色发现心房颤动患者的心房有肌溶解、心肌肥厚及广泛的间质纤维化改变。
The above results proved that polycythemia might play an important role in pathogenesis of hypoxic pulmonary hypertension and myocardial hypertrophy.
以上结果表明，红细胞增多在缺氧性肺动脉高压和右室肥大中起着重要作用。
Conclusion: Ultrasonic integrated backscatter parameters may be used to evaluate the degree of myocardial hypertrophy in the patients with hypertension.
结论：心肌背向散射参数可以用于评价高血压病患者不同肥厚程度的心肌病变。
Making myocardial mechanics researches on it contribute to bring to light the differences and connections between this two kinds of myocardial hypertrophy.
目的：为探讨长期运动训练与高血压病引起的心肌肥大在功能方面的差别，对两者进行心肌力学的分析。
Conclusions: Ultrasonic integrated backscatter parameters may be used to evaluate to the degree of myocardial hypertrophy in the patients with hypertension.
结论：心肌背向散射参数可以用于评估高血压患者心肌肥厚的不同程度。
It is suggested that with the development of myocardial hypertrophy in early stage of MI, adaptive changes in myosin ATPase activity and isoenzyme distribution occur.
结果提示：MI早期随着非梗塞心肌肥大的建立，肌球蛋白atp酶活性及其同功酶分布也发生了适应性的改变。
ConclusionBAEB has a protective effect on myocardial hypertrophy caused by Iso, and its mechanism may be related with clearance of OFR and NO concentration promotion.
结论拳参正丁醇提取物对异丙肾上腺素所致大鼠心肌肥厚具有保护作用，其作用机制与清除OFR和提高NO水平有关。
Conclusions Fosinopril could not only prevent and treat myocardial hypertrophy, but also could maintain the functions of SERCA and prevent myocardial calcium overload.
结论福辛普利不仅能防治心肌肥厚，而且能保护肌浆网钙泵功能，防止心肌钙超载损伤。
Sports Cardiology has become anew overlapping school subject is paper presents a discussion about basic research on Sports Cardiology, motional myocardial Hypertrophy;
运动心脏学已成为一门新兴的交叉学科，本文就运动心脏学的基础研究、运动性心肌肥大；
Conclusion Under chronic pressure overload, the activated ASK1 with the increasing ROS may play an important role in signal transduction pathway of myocardial hypertrophy.
结论压力超负荷下随着ROS的升高，激活的ASK1可能在心脏肥大信号传导途径中发挥重要作用。
Some of these physiological changes, in particular those relating to myocardial hypertrophy, may mimic pathological states, which are well known to pose cardiovascular risk.
生理上的某些变化，特别是与心机肥厚相关的变化会引起病理改变，从而导致心血管事件发生危险…
ConclusionTanshinone not only reverses the myocardial hypertrophy, but also reduces calcium influx and shorten action potential duration which are beneficial to prevent arrhythmia.
结论丹参酮能够在抗心肌肥厚的同时通过减少钙内流、缩短动作电位时间，起到预防心律失常的作用。
OBJECTIVE To explore the effect of simvastatin on myocardial hypertrophy and the expression of extracellular signal-regulated kinases (ERK) in spontaneously hypertensive rat (SHR).
目的探讨辛伐他汀对自发性高血压大鼠（SHR）心肌肥厚及细胞外信号调节激酶（ERK）的影响。
At present, much attention has been paid to various primary and secondary coronary artery diseases in pediatrics as well as coronary artery dysfunction due to myocardial hypertrophy.
目前儿童各类原发或继发性冠状动脉病变、心肌肥厚所导致的冠状动脉功能的改变已逐步引起广泛的关注。
Objective To observe the effects of antihypertensive agents on renin angiotensin aldosterone system, blood pressure and myocardial hypertrophy induced by high salt diet plus cold stress.
目的研究药物干预对高盐复合冷应激诱导大鼠的肾素血管紧张素醛固酮系统、血压和心肌肥厚的影响。
Objective To evaluate effect of Ramipril and Carvedilol on blood pressure, left ventricular weight, myocardial hypertrophy and left ventricular diastolic function, as well as their relations.
目的评价雷米普利、卡维地洛对高血压病左室肥厚患者的血压、左室质量、心肌肥厚消退程度及左室舒张功能的影响。
Myocardial oxidative stress and thiol-sensitive signaling molecules are implicated in pathogenesis of hypertrophy and fibrosis.
心肌氧化应激和硫醇敏感的信号转导分子参与心肌肥厚和纤维化的发病过程。
These findings suggest that aliskiren was as effective as an angiotensin receptor blocker in attenuating this measure of myocardial end-organ damage in hypertensive patients with LV hypertrophy.
这些结果表明阿里克伦在减轻高血压合并左室肥厚患者心脏终末损伤方面，与血管紧张素受体阻滞剂的效果类似。
CONCLUSION: CsA can inhibit hypertensive cardiac hypertrophy, but large dose or longer period of application will lead to severe myocardial edema, which is dose-dependent and time-dependent.
结论：环孢霉素A可抑制高血压心肌肥大，但较大剂量或较长时间应用则会产生严重心肌水肿，并且这种作用是剂量、时间依赖性的。
Objective To explore whether the Astragalus injection (AI) has effect for reversing left ventricular hypertrophy and myocardial fibrosis induced by renal vascular hypertension in rats.
目的探讨黄芪注射液对肾血管性高血压大鼠左心室肥厚及心肌纤维化有无逆转作用。
DSP and Combination use of DSP and fosinopril can prevent left ventricular fibrosis in SHR while they decrease myocardial cell apoptosis and left ventricular hypertrophy of SHR.
DSP及其与福辛普利联用降低SHR心肌细胞凋亡及减轻左室肥厚同时可减少心肌基质胶原沉积。
Conclusion QTd was decreased in exercise-induced cardiac hypertrophy subjects, which may reflect homogeneous myocardial repolarization in long term trained subjects.
结论运动诱导的心肌肥厚使QT离散度减小，反映了长期运动个体心肌复极化的稳定性。
Result: perfusion abnormality and left ventricular hypertrophy presented the image characteristics of stress myocardial perfusion imaging in 59 patients with essential hypertension.
结果：59例原发性高血压患者负荷心肌灌注显像的影像表现为灌注异常和节段性心肌肥厚，具有多壁段、多病灶的特点。
Conclusion: left ventricular hypertrophy, myocardial ischemia, left atrial enlargement and the reduction of left ventricular function can be the predictor of the cardiac arrhythmia for EH.
结论：左室肥厚、左房增大、心肌缺血、左室舒张功能减退是EH心律失常的预测因素。
The left ventricle is markedly thickened in this patient with severe hypertension that was untreated for many years. The myocardial fibers have undergone hypertrophy.
严重的高血压，并且多年未经治疗，引起左心室显著地肥厚。心肌纤维已过度肥大。
The myocardial fibers have undergone hypertrophy.
心肌纤维已过度肥大。
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