Conclusions: there is functional alteration of neuromuscular junction in ALS.
结论：肌萎缩侧索硬化存在神经肌肉接头损害。
Damage to the neuromuscular junction following prolonged retraction can also lead to muscle denervation.
持续长时间牵拉造成神经肌肉接头处的损害亦导致肌肉去神经支配。
Objective: To study the effect of Shenfu injection on fatigue in lower nerve center, neuromuscular junction and muscle.
目的：研究参附注射液对低位中枢、神经-肌接头和肌疲劳的影响。
Acting on neuromuscular junction, muscle relaxant can block excitation conduction from nerve to muscle, thus relaxing the muscle.
肌松药作用于神经肌肉接头，阻断神经至肌肉的兴奋传递。
In other words, the brain, spinal cord, cranial nerves, peripheral nerves, nerve roots, autonomic nervous system, neuromuscular junction, and muscles.
换句话说，脑、脊髓、颅神经、周围神经、神经根、自主神经系统、神经肌肉接点和肌肉。
These drugs exert their effect by inhibiting the activity of acetylcholinesterase, the enzyme that hydrolyzes acetylcholine in the neuromuscular junction.
这些药物通过抑制在神经肌肉接合部位水解乙酰胆碱的乙酰胆碱酯酶的活性而发挥效应。
The vertebrate neuromuscular junction has been very well studied, and it has long been thought that acetylcholine was the only neurotransmitter used there.
脊椎动物的神经肌肉接头已经被很好地研究，很久以来人们认为乙酰胆碱是在这里被使用的唯一神经递质。
Mice deficient in UCHL1 experience spasticity, suggesting that UCHL1 activity is required for the normal neuromuscular junction structure and function (5-7).
UCHL1缺陷的小鼠呈现痉挛状态，表明uchl1的活性对于正常结构和神经肌肉连接的功能是必须的(5 - 7)。
Objective The pathological changes on neuromuscular junction (NMJ) in the patients with myasthenia gravis (MG) or LambertEaton myasthenia syndrome (LEMS) were investigated comparatively.
目的探讨重症肌无力（MG）和癌性肌病（LEMS）时神经肌肉接头处（NMJ）的病理变化特点及其意义。
The first is that by interfering with neurotransmitter release by causing an initial facilitation, TSN eventually blocks synaptic transmission at both the neuromuscular junction and central synapses.
第一，川楝素以先易化后抑制的双相作用干扰神经递质释放，阻遏神经肌肉接头和中枢神经突触的突触传递。
These results indicate that the mutation does not interfere with the ability of agrin to induce postsynaptic structures but that it dramatically perturbs the maintenance of the neuromuscular junction.
这些结果表明，该突变不仅影响集聚蛋白诱导突触后结构的功能，也影响神经肌肉接头功能的维持。
These results indicate that the mutation does not interfere with the ability of agrin to induce postsynaptic structures but that it dramatically perturbs the maintenance of the neuromuscular junction.
这些结果表明，该突变不仅影响集聚蛋白诱导突触后结构的功能，也影响神经肌肉接头功能的维持。
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