Conclusion: Hyperglycemia for 8 weeks can change the REDOX homeostasis in liver, but can not aggravate the oxidative injury in inflammatory or fibrotic liver.
结论：8周的高血糖可能一定程度的改变肝脏的氧化还原状态，但这种改变不够明显。在并发肝脏炎症时，高血糖不能进一步加重肝脏的氧化打击。
ObjectiveTo observe the change of serum ceramide in type 2 diabetic rats and its relationship with oxidative stress.
目的观察2型糖尿病大鼠血清神经酰胺的变化及与氧化应激的关系。
OSAHS maybe through multi-ways (oxidative stress, sleep interruption) cause inflammatory reaction and adrenergic nerve excitability change to impact and induce the incidence rate of CHD increased.
OSAHS可能经氧化应激，睡眠中断引起炎症反应及交感神经兴奋性改变等多条途径影响并导致冠心病发生率增高。
EFI could change the redox status into reducing status in neonatal rat cardiomyocytes. It suggested that EFI could protect the oxidative injury of neonatal rat cardiaomyocytes induced by H2O2.
EFI可促使心肌细胞处于还原状态，对H2O2诱导的心肌细胞损伤具有保护作用。
Analysis of change of the nutrient concentration in porewater with depth indicates the relation between the oxidative-reductive conditions and the nutrient concentration.
对沉积物中孔隙水营养盐浓度随深度的变化分析，表明了沉积物孔隙水中营养盐浓度变化与氧化还原环境的关系。
Analysis of change of the nutrient concentration in porewater with depth indicates the relation between the oxidative-reductive conditions and the nutrient concentration.
对沉积物中孔隙水营养盐浓度随深度的变化分析，表明了沉积物孔隙水中营养盐浓度变化与氧化还原环境的关系。
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