AIM: To investigate the role of phosphoinositide pathway in the formation of pressure-overload cardiac hypertrophy.
目的：探讨肌醇磷脂途径在压力超负荷性心肌肥厚形成中的作用。
Suppression of target of rapamycin, a downstream effector of phosphoinositide 3-kinase, also prevented lipofuscin accumulation in the heart.
雷帕霉素作用点（即为磷酸肌醇-3激酶基因下游的受动器）的抑制也同样能够阻止脂褐质在心肌的积聚。
Objective to study the expression of phosphoinositide 3 kinase (PI3-K) and its relationship with lung cell apoptosis in rats with acute lung injury induced by oleic acid.
目的研究磷脂酰肌醇3 -激酶(PI3 - K)在油酸型急性肺损伤大鼠肺内的表达情况及其与肺组织细胞凋亡的关系。
Conclusion: Both spinal and supraspinal neuronal Phosphoinositide 3-kinase (PI3K) pathway played important role in morphine dependent and naloxone-precipitated withdrawal response in mice.
结论：脊髓和脊髓上中枢神经元pi - 3 K信号通路均参与吗啡依赖的形成及戒断反应的表达。
Conclusion: Both spinal and supraspinal neuronal Phosphoinositide 3-kinase (PI3K) pathway played important role in morphine dependent and naloxone-precipitated withdrawal response in mice.
结论：脊髓和脊髓上中枢神经元pi - 3 K信号通路均参与吗啡依赖的形成及戒断反应的表达。
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