AIM: To introduce and evaluate a canine model of secondary hypersplenism induced by splenic vein ligation.
目的：介绍并评价脾静脉结扎诱导的继发性脾功能亢进犬动物模型。
No significant changes of the leukocyte counts were observed after splenic vein ligation throughout the experiment.
脾静脉结扎后白细胞水平无显著改变。
The results showed that stable thrombocytopenia, erythropenia and splenomegaly were presented in the dogs with splenic vein ligation, and the spleen was eligible for deploying RFA.
结果表明，脾静脉结扎可以建立稳定血小板减少症和红细胞减少症，该模型适合开展脾脏射频消融治疗研究；
Conclusion The method of splenic vein ligation to induce experimental secondary hypersplenism is a simple, effective and relatively ideal model for surgical or interventional therapy in hypersplenism.
结论脾静脉结扎建立继发性功能亢进方法简单、确切，可以作为脾功能亢进外科或介入治疗的较理想模型。
A canine model of secondary hypersplenism was established by ordinal ligation of gastrosplenic vein trunk, splenic vein and its collateral branches.
通过依次结扎犬脾门处脾静脉主干、胃脾静脉干及脾静脉属枝，建立继发性脾功能亢进动物模型。
Results Splenic artery and coronary vein ligation plus C type mesocaval shunt with artificial graft was performed in 14 cases.
结果14例行脾动脉结扎、胃冠状血管及分支结扎、肠-腔C型架桥术；
ObjectiveTo investigate the efficacy and feasibility of splenic artery-vein anastomosis after hepatic artery ligation.
目的观察肝动脉阻断后脾动静脉吻合的有效性和可行性。
ObjectiveTo investigate the efficacy and feasibility of splenic artery-vein anastomosis after hepatic artery ligation.
目的观察肝动脉阻断后脾动静脉吻合的有效性和可行性。
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