PA Some partial pressure, p sub A, temperature T.
在温度T下的分压为。
Conclusions t pa and pai 1 not only participate in the pathophysiological changes of peripheral nerves after crush injury, but have some effects on peripheral nerve repair as well.
结论t PA和PAI1参与周围神经挤压伤后的病理生理改变，在损伤神经修复过程中具有一定的作用。
In group 3, the membrane was lysed within 4 24 hours after t PA injection in all cases, and no hyphema occurred.
第3组患者，4～24小时内瞳孔区的纤维蛋白膜全部吸收，无前房内出血。
Objective To assess the clinical significance of plasma t-PA, PAI-1 in acute cerebral infarction.
目的探讨血浆组织型纤溶酶原激活物(t - PA)及其抑制物(PAI - 1)在急性脑梗死发病中的意义。
The key safety end point was severe or moderate bleeding according to the Global Utilization of Streptokinase and t-PA for Occluded Coronary Arteries (GUSTO) definition.
首要效力观察终点为基于“通用链激酶与t-PA针对冠脉闭塞（GUSTO）”定义的中度或重度的出血；
Results: The changes of high TG, PAI-1 and low level t-pA were most significant abnormal charctor of Cerbral Infarction.
结果：血高TG、高PAI 1，与低t PA含量变化为脑梗塞患者异常变化特征，与正常组相比有显著性差异（P。
The study compared the efficacy and safety of ultrasound with t-PA vs t-PA only in a trial involving 126 patients.
该研究比较了联合应用超声波和t - PA与单独使用t - PA的作用与安全性，研究涉及了126例患者。
The thrombolytic effect was compared between urokinase (UK) and human tissue plasminogen activator (t-PA) in rabbits with experimental iliofemoral artery thrombus.
本文用尿激酶(UK)和人体组织型纤溶酶原激活剂(t - PA)进行了兔髂股动脉血栓实验性溶解的对照研究。
Therefore, t-PA is considered as a more effective and safer thrombolytic agent.
故t-PA是一种效果较好、治疗剂量时较为安全的溶栓剂。
Objective the effect of vascular smooth muscle cells (VSMCs) on the abilities of secreting t-PA and PAI-1 by th cultured endothelial cells (ECs) under shear stress has been studied.
目的探讨切应力作用下联合培养的血管平滑肌细胞对内皮细胞分泌t - PA和PAI- 1的影响。
Results: The activity of PAI-1 was higher and t-PA was lower in patients with coronary heart disease, and this difference is also found between Type a and Non Type a patients.
结果：冠心病患者比正常人群的PAI - 1活性上升，t - PA活性下降；这种改变在不同行为类型之间比较，A型行为类型比非a型行为类型的冠心病患者PAI - 1的活性升高而t - PA活性降低。
Objective To investigate effects of fibrinolytic system upon deep venous thrombosis, focused on PLG, t-PA and PAI-1.
目的以纤溶酶原、t - PA和PAI - 1为轴心，探讨纤溶系统对深静脉血栓形成(DVT)的影响。
The morphology had relationship with t-PA production.
菌体形态和t-PA的产生密切相关。
Conclusion the unbalance between t-PA and PAI can promote the formation of micro-thrombus and further exacerbate high pulmonary pressure.
结论t - PA及PAI关系失衡，有助于动脉内微血栓形成，促进或加重肺动脉高压的形成。
The purified t-PA has a higher affinity for fibrin than urokinase. In addition, the melanoma plasminogen activator appeared to be immunologically different from urokinase.
纯化的t-PA与尿激酶相比较，发现前者有更高亲和纤维蛋白的能力。
According to reports, Nattokinase has plasmin-like bio-characteristic that lyses fibrin directly, or increases activity of pro-urokinase and t-PA (Tissue Plasminogen Activator).
据报道，纳豆激酶有和胞浆素一样的可直接分解纤维蛋白的生物特性，或者增强尿激酶原和t—PA(人组织型纤溶酶原激活剂)的活性。
According to reports, Nattokinase has plasmin-like bio-characteristic that lyses fibrin directly, or increases activity of pro-urokinase and t-PA (Tissue Plasminogen Activator).
据报道，纳豆激酶有和胞浆素一样的可直接分解纤维蛋白的生物特性，或者增强尿激酶原和t—PA(人组织型纤溶酶原激活剂)的活性。
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