It shows the role of TNF-alpha and its receptors in the production of beta-1,4 galactosyltransferase I and V mRNAs by rat primary astrocytes.
这显示了巢蛋白和阶段特异性胚胎抗原-1在大鼠2型星形胶质细胞中的表达。
In this experiment, the serum TNF - a.
在本实验中，应用尿激酶后血清tnf一。
TNF and insulin levels were highly correlated.
肿瘤坏死因子与胰岛素水平有强相关性。
Remicade belongs to a class of drugs called tumor necrosis factor (TNF) blockers.
Remicade属于被称为肿瘤坏死因子(TNF)阻断剂类药物。
TNF-a and NO are two major tumorcidal effector molecules of activated macrophages.
及NO是激活的巨噬细胞杀伤肿瘤细胞的主要效应分子。
Then, a fusion protein expression system was used to express the TNF half molecule.
用融合蛋白表达系统对该半分子进行大肠杆菌表达。
SIR is relative to inducing and expression of some of cytokines such as ET and TNF .
全身炎症反应与一些细胞因子如内皮素、肿瘤坏死因子等的诱导表达有关。
Conclusion: Reactive oxygen intermediates was a mediator of apoptosis induced by TNF.
结论：活性氧中间产物是TNF诱导细胞凋亡的中介体之一。
Objective To study the changes of serum TNF and SA in patients with rheumatic arthritis.
目的探讨风湿性关节炎患者血清中TNF和SA含量的变化。
Aim to study the effects of TNF receptor blocking peptide on adjuvant arthritis in rats.
目的观察肿瘤坏死因子(TNF)受体封闭肽对大鼠佐剂性关节炎的影响。
TNF induced in the cell culture was demonstrated by monoclone antibody neutralizing test.
诱生出的TNF通过单克隆抗体中和试验得到确证。
Conclusion: TNF gene may be correlated with the susceptibility of thymoma and thymoma with MG.
结论TNF基因多态性与胸腺瘤发病及胸腺瘤发生重症肌无力相关。
The changes of CEA and TNF in the pleural effusion were examined before and after the treatment.
并检测治疗前后胸水中癌胚抗原和肿瘤坏死因子的改变。
Patients who develop a fungal infection may be advised to stop the TNF blocker until they recover.
受到真菌感染的患者可能会被建议在真菌疾病康复前中止使用TNF阻滞剂。
Objective To study the effect of ischemic preconditioning on the apoptosis and the TNF plasma level.
目的探讨缺血预处理对心肌细胞凋亡及血浆TNF水平的影响。
Lower levels of TNF and higher levels of SOD are apparently beneficial to wound healing after trauma.
内源性低浓度tnf及抗氧化能力的提高，对创面修复是有益的。
Tumor necrosis factor (TNF) is a potent cytokine that elicits a broad spectrum of biological responses.
肿瘤坏死因子tnf是一种多功能的细胞因子，具有广泛的生物学活性。
Objective To investigate the clinical significance of the changes TNF and NPY in ovary cancer patients.
目的探讨血清细胞因子、血浆NPY术前术后的变化对卵巢癌患者的临床意义。
Patients taking TNF blockers should be aware that they are more susceptible to serious fungal infections.
使用TNF阻滞剂的患者应该注意到他们更易受到严重的真菌感染。
A proliferation-induced ligand(APRIL)is a recently found member of tumor necrosis factor (TNF)superfamily.
增殖诱导配体（APRIL）是近年来发现的肿瘤坏死因子（TNF）超家族成员。
Objective To study the relationship between nitric oxide(NO), tumor necrosis factor(TNF) and acute pancreatitis.
目的探讨一氧化氮（NO）和肿瘤坏死因子（TNF）在急性胰腺炎中的作用。
Conclusion the plasma TNF level closely correlates with the complicated infectious shock and with the organ failure.
结论血浆TNF水平与并发感染性休克或器官功能衰竭密切相关。
We hypothesized that TNF induces opposing inflammatory and remodeling responses in HF that are TNF-receptor (TNFR) specific.
我们的研究假设是TNF通过特异的TNF受体(TNFR)诱导相反的炎症和重构反应。
The FDA also reviewed 69 cases of psoriasis and said it found a possible link between the skin disorder and use of TNF blockers.
FDA也检查了69例牛皮癣，称皮肤病症和TNF拮抗剂之间可能存在某种的联系。
Methods The serum content of TNF-a was detected by radioimmunoassay (RIA)in 20 healthy controls and 70 patients with malignant tumor.
方法应用放射免疫测定法对20例健康对照者和70例肿瘤患者进行了血清TNF-a含量测定。
Objective To study changes of NO, ET, TNF content in blood in rats with focal cerebral ischemia reperfusion injury treated by Aspirin.
目的探讨阿司匹林对大鼠局灶性脑缺血再灌注损伤中血一氧化氮（NO）、内皮素（ET）、肿瘤坏死因子（TNF）含量的影响。
Objective to study the protective effect of removing circulatory TNF by immunoadsorption on endotoxin induced acute lung injury in rabbits.
目的研究亲和免疫吸附清除循环肿瘤坏死因子(TNF)对内毒素性急性肺损伤动物的保护作用。
The FDA also has received reports of cases of coccidioidomycosis and blastomycosis, including deaths, in patients treated with TNF blockers.
还收到一些使用TNF阻滞剂导致球孢子病和芽生菌病的报告，甚至还有死亡病例。
Additional required updates to the prescribing information include incorporation of reports of psoriasis associated with the use of TNF blockers.
要求更新的其他处方信息包括纳入与TNF阻滞剂使用相关的银屑病报告。
The first TRAFs identified, TRAF1 and TRAF2, were found by virtue of their interactions with the cytoplasmic domain of TNF-receptor 2 (TNFRII) (4).
首先鉴定出的TRAF家族成员是TRAF1和TRAF2，发现它们能够与TNF受体2 (TNFRII)的胞质结构域相互作用(4)。
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