The pathological changes of tunica media were reversible, without vasomotion and play a stent role only.
中膜层病理学改变是可逆性的，但已失去血管舒缩功能，仅具生物支架作用。
The microstructure component in tunica media of umbilical cord vein were measured by the computer image analysis system.
光镜观察后，用计算机图像分析系统测量其中膜显微结构成分的相对含量。
Methods Found the relationship of aortic dissection false lumens ectonexine of aortic tunica media by test and measurement their shape-changed strength ratio.
方法通过胸主动脉夹层假腔内外层的屈服形状改变比能，发现两者间的关系。结果60%的实验犬形成夹层模型。
MMP-2 protein expression can be observed in atherosclerosis, mainly in macrophage, foam cells, endothelial cell, smooth muscle cell in endomembrane, smooth muscle cell and fibrocyre in tunica media.
MMP-2在粥样硬化冠脉斑块中有表达，表达的阳性部位为内膜的血管内皮细胞胞浆、巨噬细胞胞浆、平滑肌细胞胞浆和中膜的平滑肌细胞胞浆。
During atherogenesis, vascular smooth muscle cells proliferate, migrate from tunica intima to media and uptake lipid then become foam cells.
我们推测动脉粥样硬化形成过程中血管平滑肌细胞能量代谢发生了改变。
During atherogenesis, vascular smooth muscle cells proliferate, migrate from tunica intima to media and uptake lipid then become foam cells.
我们推测动脉粥样硬化形成过程中血管平滑肌细胞能量代谢发生了改变。
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