Activation of the FSH receptor is known to contribute to the signaling of vascular endothelial growth factor (VEGF), a protein that stimulates the growth of blood vessels, including those in tumors.
现已知fsh受体的激活可以促进血管内皮生长因子(VEGF)的信号传导通路，而VEGF可以促进包括肿瘤组织在内的血管的生长。
Vascular endothelial growth factor (VEGF) and its receptor play a critical role in tumor-associated angiogenesis and have become the new targets of anti-tumor therapy.
血管内皮细胞生长因子(VEGF)及其受体在肿瘤血管新生中起重要作用，阻断VEGF的生成已成为抗肿瘤治疗的新靶点。
Vascular endothelial growth factor (VEGF) and its receptor play a critical role in tumor associated angiogenesis and has become the new targets of anti tumor therapy.
血管内皮细胞生长因子及其受体在肿瘤新生血管形成中起重要作用，成为肿瘤治疗的新靶点。
Bevacizumab blocks the action of the vascular endothelial growth factor (VEGF) receptor, a key factor in tumor angiogenesis, growth, and metastasis.
贝伐单抗阻断血管内皮生长因子(VEGF)受体活性，是肿瘤血管生成，增殖和转移的重要因子。
Bevacizumab blocks the action of the vascular endothelial growth factor (VEGF) receptor, a key factor in tumor angiogenesis, growth, and metastasis.
贝伐单抗阻断血管内皮生长因子(VEGF)受体活性，是肿瘤血管生成，增殖和转移的重要因子。
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