Hemorheology change was the physiological basis of that inhibitory action to vascular intimal hyperplasia.
血液流变学的变化是大黄抑制血管搭桥术后血管内膜增生的生理学基础。
CONCLUSION: Local transfection of VEGF165 gene restrains intimal hyperplasia and restenosis of vessels, which lays a foundation for future gene therapy of vascular intimal hyperplasia.
结论：局部转染vegf165基因可抑制血管新生内膜增生及血管再狭窄，为将来血管内膜增生的基因治疗奠定基础。
AIM: To explore the role of intimal hyperplasia and vascular remodeling in the formation of vascular restenosis.
目的：探讨在血管再狭窄形成过程中内膜增生及血管重塑的作用。
The major complication of vascular recanalization is intimal hyperplasia which in due mainly to over-proliferation and migration of smooth muscle cells(SMC).
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是术后血管再通的主要障碍。
The major complication of vascular recanalization is intimal hyperplasia which in duc mainly to over-proliferation and migration of smooth muscle cells (SMC).
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是血管成形术后再狭窄的重要原因之一。
The major complication of vascular recanalization is intimal hyperplasia which in duc mainly to over-proliferation and migration of smooth muscle cells (SMC).
血管平滑肌细胞过度增殖及游移所致的血管内膜增生是血管成形术后再狭窄的重要原因之一。
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